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D C Huang

Showing results (31-40 of 46) with videos related to

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The Journal of Cell Biology|February 23, 2000
Debcl, a proapoptotic Bcl-2 homologue, is a component of the Drosophila melanogaster cell death machineryP A Colussi, L M Quinn, D C Huang, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 28, 1999
Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L)D C Huang, M Hahne, M Schroeter, et al.
Annals of the New York Academy of Sciences|February 24, 2001
The role of the pro-apoptotic Bcl-2 family member bim in physiological cell deathP Bouillet, D C Huang, L A O'Reilly, et al.
The Journal of Cell Biology|May 3, 2000
Pro-apoptotic apoptosis protease-activating factor 1 (Apaf-1) has a cytoplasmic localization distinct from Bcl-2 or Bcl-x(L)G Hausmann, L A O'Reilly, R van Driel, et al.
Science (New York, N.Y.)|September 8, 2001
Bmf: a proapoptotic BH3-only protein regulated by interaction with the myosin V actin motor complex, activated by anoikisH Puthalakath, A Villunger, L A O'Reilly, et al.
The American Journal of Pathology|August 10, 2000
The proapoptotic BH3-only protein bim is expressed in hematopoietic, epithelial, neuronal, and germ cellsL A O'Reilly, L Cullen, J Visvader, et al.
Science (New York, N.Y.)|November 27, 1999
Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunityP Bouillet, D Metcalf, D C Huang, et al.
Annals of the New York Academy of Sciences|March 28, 2001
The role of bim, a proapoptotic BH3-only member of the Bcl-2 family in cell-death controlA Strasser, H Puthalakath, P Bouillet, et al.
Stroke|March 1, 1987
Intravenous prostacyclin in acute nonhemorrhagic stroke: a placebo-controlled double-blind trialC Y Hsu, R E Faught, A J Furlan, et al.
International Immunology|December 23, 1999
Transgenic overexpression of human Bcl-2 in islet beta cells inhibits apoptosis but does not prevent autoimmune destructionJ Allison, H Thomas, D Beck, et al.
Pageof 5

Showing results (31-40 of 46) with videos related to

Sort By:
Pageof 5
The Journal of Cell Biology|February 23, 2000
Debcl, a proapoptotic Bcl-2 homologue, is a component of the Drosophila melanogaster cell death machineryP A Colussi, L M Quinn, D C Huang, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 28, 1999
Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L)D C Huang, M Hahne, M Schroeter, et al.
Annals of the New York Academy of Sciences|February 24, 2001
The role of the pro-apoptotic Bcl-2 family member bim in physiological cell deathP Bouillet, D C Huang, L A O'Reilly, et al.
The Journal of Cell Biology|May 3, 2000
Pro-apoptotic apoptosis protease-activating factor 1 (Apaf-1) has a cytoplasmic localization distinct from Bcl-2 or Bcl-x(L)G Hausmann, L A O'Reilly, R van Driel, et al.
Science (New York, N.Y.)|September 8, 2001
Bmf: a proapoptotic BH3-only protein regulated by interaction with the myosin V actin motor complex, activated by anoikisH Puthalakath, A Villunger, L A O'Reilly, et al.
The American Journal of Pathology|August 10, 2000
The proapoptotic BH3-only protein bim is expressed in hematopoietic, epithelial, neuronal, and germ cellsL A O'Reilly, L Cullen, J Visvader, et al.
Science (New York, N.Y.)|November 27, 1999
Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunityP Bouillet, D Metcalf, D C Huang, et al.
Annals of the New York Academy of Sciences|March 28, 2001
The role of bim, a proapoptotic BH3-only member of the Bcl-2 family in cell-death controlA Strasser, H Puthalakath, P Bouillet, et al.
Stroke|March 1, 1987
Intravenous prostacyclin in acute nonhemorrhagic stroke: a placebo-controlled double-blind trialC Y Hsu, R E Faught, A J Furlan, et al.
International Immunology|December 23, 1999
Transgenic overexpression of human Bcl-2 in islet beta cells inhibits apoptosis but does not prevent autoimmune destructionJ Allison, H Thomas, D Beck, et al.
Pageof 5