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The Journal of Biological Chemistry|February 12, 1998
Suppression of neuronal and cardiac transient outward currents by viral gene transfer of dominant-negative Kv4.2 constructsD C Johns, H B Nuss, E MarbanAdvances in Experimental Medicine and Biology|January 1, 1995
Prospects for genetic manipulation of cardiac excitabilityJ H Lawrence, D C Johns, N Chiamvimonvat, et al.The Journal of Physiology|September 1, 1994
Electrophysiological properties of neonatal mouse cardiac myocytes in primary cultureH B Nuss, E MarbanThe Journal of Clinical Investigation|August 1, 1995
Adenovirus-mediated expression of a voltage-gated potassium channel in vitro (rat cardiac myocytes) and in vivo (rat liver). A novel strategy for modifying excitabilityD C Johns, H B Nuss, N Chiamvimonvat, et al.Circulation|March 12, 1998
Phenotypic characterization of a novel long-QT syndrome mutation (R1623Q) in the cardiac sodium channelN G Kambouris, H B Nuss, D C Johns, et al.Gene Therapy|October 1, 1996
Reversal of potassium channel deficiency in cells from failing hearts by adenoviral gene transfer: a prototype for gene therapy for disorders of cardiac excitability and contractilityH B Nuss, D C Johns, S Kääb, et al.The Journal of Clinical Investigation|December 15, 1996
Local anesthetics as effectors of allosteric gating. Lidocaine effects on inactivation-deficient rat skeletal muscle Na channelsJ R Balser, H B Nuss, D W Orias, et al.The Journal of Clinical Investigation|March 17, 1999
Overexpression of a human potassium channel suppresses cardiac hyperexcitability in rabbit ventricular myocytesH B Nuss, E Marbán, D C JohnsThe Journal of General Physiology|May 1, 1996
Functional consequences of lidocaine binding to slow-inactivated sodium channelsJ R Balser, H B Nuss, D N Romashko, et al.Proceedings of the National Academy of Sciences of the United States of America|April 29, 1997
Ultrarapid, highly efficient viral gene transfer to the heartJ K Donahue, K Kikkawa, D C Johns, et al.Pageof 17