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D D Chang

Showing results (11-20 of 27) with videos related to

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Molecular and Cellular Biology|September 1, 1986
Precise assignment of the heavy-strand promoter of mouse mitochondrial DNA: cognate start sites are not required for transcriptional initiationD D Chang, D A Clayton
Molecular and Cellular Biology|September 1, 1986
Precise assignment of the light-strand promoter of mouse mitochondrial DNA: a functional promoter consists of multiple upstream domainsD D Chang, D A Clayton
Journal of Cellular Biochemistry|November 14, 2000
Overexpression of wild-type RhoA produces growth arrest by disrupting actin cytoskeleton and microtubulesY Song, C Wong, D D Chang
Biochimica Et Biophysica Acta|July 14, 1987
Roles for a promoter and RNA processing in the synthesis of mitochondrial displacement-loop strandsD D Chang, R P Fisher, D A Clayton
Molecular and Cellular Biology|January 1, 1986
Minor transcription initiation events indicate that both human mitochondrial promoters function bidirectionallyD D Chang, J E Hixson, D A Clayton
The EMBO Journal|June 1, 1985
Replication priming and transcription initiate from precisely the same site in mouse mitochondrial DNAD D Chang, W W Hauswirth, D A Clayton
Gene|February 17, 2001
Characterization of mouse epithelial protein lost in neoplasm (EPLIN) and comparison of mammalian and zebrafish EPLINR S Maul, C Sachi Gerbin, D D Chang
The Journal of Cell Biology|September 8, 1997
ICAP-1, a novel beta1 integrin cytoplasmic domain-associated protein, binds to a conserved and functionally important NPXY sequence motif of beta1 integrinD D Chang, C Wong, H Smith, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 1, 1991
Structural analysis of the interaction between the human immunodeficiency virus Rev protein and the Rev response elementJ Kjems, M Brown, D D Chang, et al.
Gene|May 12, 2000
Characterization of the human EPLIN (Epithelial Protein Lost in Neoplasm) gene reveals distinct promoters for the two EPLIN isoformsS Chen, R S Maul, H R Kim, et al.
Pageof 3

Showing results (11-20 of 27) with videos related to

Sort By:
Pageof 3
Molecular and Cellular Biology|September 1, 1986
Precise assignment of the heavy-strand promoter of mouse mitochondrial DNA: cognate start sites are not required for transcriptional initiationD D Chang, D A Clayton
Molecular and Cellular Biology|September 1, 1986
Precise assignment of the light-strand promoter of mouse mitochondrial DNA: a functional promoter consists of multiple upstream domainsD D Chang, D A Clayton
Journal of Cellular Biochemistry|November 14, 2000
Overexpression of wild-type RhoA produces growth arrest by disrupting actin cytoskeleton and microtubulesY Song, C Wong, D D Chang
Biochimica Et Biophysica Acta|July 14, 1987
Roles for a promoter and RNA processing in the synthesis of mitochondrial displacement-loop strandsD D Chang, R P Fisher, D A Clayton
Molecular and Cellular Biology|January 1, 1986
Minor transcription initiation events indicate that both human mitochondrial promoters function bidirectionallyD D Chang, J E Hixson, D A Clayton
The EMBO Journal|June 1, 1985
Replication priming and transcription initiate from precisely the same site in mouse mitochondrial DNAD D Chang, W W Hauswirth, D A Clayton
Gene|February 17, 2001
Characterization of mouse epithelial protein lost in neoplasm (EPLIN) and comparison of mammalian and zebrafish EPLINR S Maul, C Sachi Gerbin, D D Chang
The Journal of Cell Biology|September 8, 1997
ICAP-1, a novel beta1 integrin cytoplasmic domain-associated protein, binds to a conserved and functionally important NPXY sequence motif of beta1 integrinD D Chang, C Wong, H Smith, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 1, 1991
Structural analysis of the interaction between the human immunodeficiency virus Rev protein and the Rev response elementJ Kjems, M Brown, D D Chang, et al.
Gene|May 12, 2000
Characterization of the human EPLIN (Epithelial Protein Lost in Neoplasm) gene reveals distinct promoters for the two EPLIN isoformsS Chen, R S Maul, H R Kim, et al.
Pageof 3