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D E Michele

Showing results (1-10 of 14) with videos related to

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Trends in Cardiovascular Medicine|March 10, 2001
Contractile dysfunction in hypertrophic cardiomyopathy: elucidating primary defects of mutant contractile proteins by gene transferD E Michele, J M Metzger
Journal of Molecular Medicine (Berlin, Germany)|February 24, 2001
Physiological consequences of tropomyosin mutations associated with cardiac and skeletal myopathiesD E Michele, J M Metzger
Biochimie|July 1, 1997
Vanadates form insoluble complexes with histonesD E Michele, D Thomsen, L L Louters
The Journal of Clinical Investigation|December 10, 1999
A nemaline myopathy mutation in alpha-tropomyosin causes defective regulation of striated muscle force productionD E Michele, F P Albayya, J M Metzger
Biochimie|January 1, 1996
Histone H4 stimulates glucose uptake through the insulin receptorL L Louters, D E Michele, J D Pearson
The Journal of Cell Biology|June 29, 1999
Thin filament protein dynamics in fully differentiated adult cardiac myocytes: toward a model of sarcomere maintenanceD E Michele, F P Albayya, J M Metzger
Proceedings of the National Academy of Sciences of the United States of America|October 16, 1999
Parvalbumin gene transfer corrects diastolic dysfunction in diseased cardiac myocytesP A Wahr, D E Michele, J M Metzger
Nature Medicine|December 2, 1999
Direct, convergent hypersensitivity of calcium-activated force generation produced by hypertrophic cardiomyopathy mutant alpha-tropomyosins in adult cardiac myocytesD E Michele, F P Albayya, J M Metzger
American Journal of Physiology. Heart and Circulatory Physiology|August 3, 2000
Effects of aging on single cardiac myocyte function in Fischer 344 x Brown Norway ratsP A Wahr, D E Michele, J M Metzger
Circulation Research|June 12, 1999
Effects of myosin heavy chain isoform switching on Ca2+-activated tension development in single adult cardiac myocytesJ M Metzger, P A Wahr, D E Michele, et al.
Pageof 2

Showing results (1-10 of 14) with videos related to

Sort By:
Pageof 2
Trends in Cardiovascular Medicine|March 10, 2001
Contractile dysfunction in hypertrophic cardiomyopathy: elucidating primary defects of mutant contractile proteins by gene transferD E Michele, J M Metzger
Journal of Molecular Medicine (Berlin, Germany)|February 24, 2001
Physiological consequences of tropomyosin mutations associated with cardiac and skeletal myopathiesD E Michele, J M Metzger
Biochimie|July 1, 1997
Vanadates form insoluble complexes with histonesD E Michele, D Thomsen, L L Louters
The Journal of Clinical Investigation|December 10, 1999
A nemaline myopathy mutation in alpha-tropomyosin causes defective regulation of striated muscle force productionD E Michele, F P Albayya, J M Metzger
Biochimie|January 1, 1996
Histone H4 stimulates glucose uptake through the insulin receptorL L Louters, D E Michele, J D Pearson
The Journal of Cell Biology|June 29, 1999
Thin filament protein dynamics in fully differentiated adult cardiac myocytes: toward a model of sarcomere maintenanceD E Michele, F P Albayya, J M Metzger
Proceedings of the National Academy of Sciences of the United States of America|October 16, 1999
Parvalbumin gene transfer corrects diastolic dysfunction in diseased cardiac myocytesP A Wahr, D E Michele, J M Metzger
Nature Medicine|December 2, 1999
Direct, convergent hypersensitivity of calcium-activated force generation produced by hypertrophic cardiomyopathy mutant alpha-tropomyosins in adult cardiac myocytesD E Michele, F P Albayya, J M Metzger
American Journal of Physiology. Heart and Circulatory Physiology|August 3, 2000
Effects of aging on single cardiac myocyte function in Fischer 344 x Brown Norway ratsP A Wahr, D E Michele, J M Metzger
Circulation Research|June 12, 1999
Effects of myosin heavy chain isoform switching on Ca2+-activated tension development in single adult cardiac myocytesJ M Metzger, P A Wahr, D E Michele, et al.
Pageof 2