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D E Moller

Showing results (31-40 of 92) with videos related to

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Endocrinology|March 1, 1993
Ligand-binding properties of the two isoforms of the human insulin receptorY Yamaguchi, J S Flier, H Benecke, et al.
The American Journal of Physiology|February 1, 1994
Human rsk isoforms: cloning and characterization of tissue-specific expressionD E Moller, C H Xia, W Tang, et al.
Biochemical and Biophysical Research Communications|October 14, 1996
Regulation of acetyl coenzyme-A carboxylase gene in a transgenic animal modelD Ekhterae, H J Tae, S Daniel, et al.
The Journal of Biological Chemistry|December 15, 1995
Insulin stimulation of mitogen-activated protein kinase, p90rsk, and p70 S6 kinase in skeletal muscle of normal and insulin-resistant mice. Implications for the regulation of glycogen synthaseP Y Chang, Y Le Marchand-Brustel, L A Cheatham, et al.
The Journal of Biological Chemistry|September 5, 1990
A naturally occurring mutation of insulin receptor alanine 1134 impairs tyrosine kinase function and is associated with dominantly inherited insulin resistanceD E Moller, A Yokota, M F White, et al.
Diabetes Research and Clinical Practice|August 1, 1995
Insulin-like growth factor I (rhIGF-I) as a therapeutic agent for hyperinsulinemic insulin-resistant diabetes mellitusA C Moses, L A Morrow, M O'Brien, et al.
Diabetologia|May 1, 1992
Insulin receptor and insulin-responsive glucose transporter (GLUT 4) mutations and polymorphisms in a Welsh type 2 (non-insulin-dependent) diabetic populationS O'Rahilly, A Krook, R Morgan, et al.
The Journal of Biological Chemistry|May 26, 1995
Impaired insulin signaling in skeletal muscles from transgenic mice expressing kinase-deficient insulin receptorsP Y Chang, L J Goodyear, H Benecke, et al.
The Journal of Clinical Endocrinology and Metabolism|February 1, 1989
Octreotide suppresses both growth hormone (GH) and GH-releasing hormone (GHRH) in acromegaly due to ectopic GHRH secretionD E Moller, A C Moses, K Jones, et al.
Biochemical and Biophysical Research Communications|October 15, 1993
The stimulation of pp42mapkinase by insulin does not correlate with its metabolic actions in cells overexpressing mutant insulin receptorsL Pang, D F Lazar, D E Moller, et al.
Pageof 10

Showing results (31-40 of 92) with videos related to

Sort By:
Pageof 10
Endocrinology|March 1, 1993
Ligand-binding properties of the two isoforms of the human insulin receptorY Yamaguchi, J S Flier, H Benecke, et al.
The American Journal of Physiology|February 1, 1994
Human rsk isoforms: cloning and characterization of tissue-specific expressionD E Moller, C H Xia, W Tang, et al.
Biochemical and Biophysical Research Communications|October 14, 1996
Regulation of acetyl coenzyme-A carboxylase gene in a transgenic animal modelD Ekhterae, H J Tae, S Daniel, et al.
The Journal of Biological Chemistry|December 15, 1995
Insulin stimulation of mitogen-activated protein kinase, p90rsk, and p70 S6 kinase in skeletal muscle of normal and insulin-resistant mice. Implications for the regulation of glycogen synthaseP Y Chang, Y Le Marchand-Brustel, L A Cheatham, et al.
The Journal of Biological Chemistry|September 5, 1990
A naturally occurring mutation of insulin receptor alanine 1134 impairs tyrosine kinase function and is associated with dominantly inherited insulin resistanceD E Moller, A Yokota, M F White, et al.
Diabetes Research and Clinical Practice|August 1, 1995
Insulin-like growth factor I (rhIGF-I) as a therapeutic agent for hyperinsulinemic insulin-resistant diabetes mellitusA C Moses, L A Morrow, M O'Brien, et al.
Diabetologia|May 1, 1992
Insulin receptor and insulin-responsive glucose transporter (GLUT 4) mutations and polymorphisms in a Welsh type 2 (non-insulin-dependent) diabetic populationS O'Rahilly, A Krook, R Morgan, et al.
The Journal of Biological Chemistry|May 26, 1995
Impaired insulin signaling in skeletal muscles from transgenic mice expressing kinase-deficient insulin receptorsP Y Chang, L J Goodyear, H Benecke, et al.
The Journal of Clinical Endocrinology and Metabolism|February 1, 1989
Octreotide suppresses both growth hormone (GH) and GH-releasing hormone (GHRH) in acromegaly due to ectopic GHRH secretionD E Moller, A C Moses, K Jones, et al.
Biochemical and Biophysical Research Communications|October 15, 1993
The stimulation of pp42mapkinase by insulin does not correlate with its metabolic actions in cells overexpressing mutant insulin receptorsL Pang, D F Lazar, D E Moller, et al.
Pageof 10