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The Journal of Biological Chemistry|February 13, 1999
c-Jun is a JNK-independent coactivator of the PU.1 transcription factorG Behre, A J Whitmarsh, M P Coghlan, et al.Blood|May 15, 1995
Neutrophils and monocytes express high levels of PU.1 (Spi-1) but not Spi-BH M Chen, P Zhang, M T Voso, et al.Oncogene|October 19, 1995
PU.1 (Spi-1) autoregulates its expression in myeloid cellsH Chen, D Ray-Gallet, P Zhang, et al.Gene|March 25, 1998
Two promoters direct expression of the murine Spi-B gene, an Ets family transcription factorH M Chen, D A Gonzalez, H S Radomska, et al.Molecular and Cellular Biology|March 1, 1996
CCAAT enhancer-binding protein (C/EBP) and AML1 (CBF alpha2) synergistically activate the macrophage colony-stimulating factor receptor promoterD E Zhang, C J Hetherington, S Meyers, et al.Leukemia|July 9, 2016
Restoration of MYC-repressed targets mediates the negative effects of GM-CSF on RUNX1-ETO leukemogenicityS Weng, S Matsuura, C T Mowery, et al.Blood|September 9, 2000
Analysis of the role of AML1-ETO in leukemogenesis, using an inducible transgenic mouse modelK L Rhoades, C J Hetherington, N Harakawa, et al.Proceedings of the National Academy of Sciences of the United States of America|August 30, 2001
AML1-ETO expression is directly involved in the development of acute myeloid leukemia in the presence of additional mutationsY Yuan, L Zhou, T Miyamoto, et al.Nature Genetics|March 1, 1997
Embryonic lethality and impairment of haematopoiesis in mice heterozygous for an AML1-ETO fusion geneD A Yergeau, C J Hetherington, Q Wang, et al.Genes and Immunity|May 9, 2014
Contribution of increased ISG15, ISGylation and deregulated type I IFN signaling in Usp18 mutant mice during the course of bacterial infectionsS M Dauphinee, E Richer, M M Eva, et al.Pageof 6