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Anesthesiology|October 1, 1994
Repetitive rapid increases in desflurane concentration blunt transient cardiovascular stimulation in humansR B Weiskopf, E I Eger, M Noorani, et al.
Anesthesia and Analgesia|June 1, 1991
Absorption and degradation of sevoflurane and isoflurane in a conventional anesthetic circuitJ Liu, M J Laster, E I Eger, et al.
Anesthesiology|December 1, 1995
Cardiovascular stimulation induced by rapid increases in desflurane concentration in humans results from activation of tracheopulmonary and systemic receptorsR B Weiskopf, E I Eger, M Daniel, et al.
Anesthesia and Analgesia|June 1, 1993
Electrical stimulation as a substitute for the tail clamp in the determination of minimum alveolar concentrationM J Laster, J Liu, E I Eger, et al.
Anesthesia and Analgesia|July 1, 1992
Isoflurane anesthesia prevents unconscious learningR Dwyer, H L Bennett, E I Eger, et al.
Anesthesia and Analgesia|December 8, 1998
Ethanol concentrations approaching minimum alveolar anesthetic concentration are required to suppress learning in a fear-potentiated startle paradigm in ratsJ M Sonner, J Li, L Kandel, et al.
The Journal of Pharmacology and Experimental Therapeutics|November 1, 1975
The effect of temperature on a d-tubocurarine neuromuscular blockade and its antagonism by neostigmineR D Miller, L S Van Nyhis, E I Eger
Anesthesiology|February 3, 1998
Fentanyl augments the blockade of the sympathetic response to incision (MAC-BAR) produced by desflurane and isoflurane: desflurane and isoflurane MAC-BAR without and with fentanylM Daniel, R B Weiskopf, M Noorani, et al.
Anesthesiology|January 1, 1996
Propofol fails to attenuate the cardiovascular response to rapid increases in desflurane concentrationM Daniel, E I Eger, R B Weiskopf, et al.
Anesthesia and Analgesia|January 3, 2001
Neither GABA(A) nor strychnine-sensitive glycine receptors are the sole mediators of MAC for isofluraneY Zhang, S Wu, E I Eger, et al.
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