Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

D G Kay

Showing results (1-10 of 17) with videos related to

Pageof 2
Sort By:
Journal of Virology|November 1, 1993
Identification of the infected target cell type in spongiform myeloencephalopathy induced by the neurotropic Cas-Br-E murine leukemia virusC Gravel, D G Kay, P Jolicoeur
Journal of Virology|December 1, 1996
The prion protein gene is dispensable for the development of spongiform myeloencephalopathy induced by the neurovirulent Cas-Br-E murine leukemia virusP Jolicoeur, G Massé, D G Kay
Journal of Bacteriology|March 1, 1980
Ornithine decarboxylase activity and cell cycle regulation in Saccharomyces cerevisiaeD G Kay, R A Singer, G C Johnston
Proceedings of the National Academy of Sciences of the United States of America|February 15, 1991
Retrovirus-induced spongiform myeloencephalopathy in mice: regional distribution of infected target cells and neuronal loss occurring in the absence of viral expression in neuronsD G Kay, C Gravel, Y Robitaille, et al.
Journal of Virology|December 1, 1991
The majority of cells infected with the defective murine AIDS virus belong to the B-cell lineageM Huang, C Simard, D G Kay, et al.
Journal of Virology|August 1, 1990
Substitution of the U3 long terminal repeat region of the neurotropic Cas-Br-E retrovirus affects its disease-inducing potentialY Paquette, D G Kay, E Rassart, et al.
Biochemical and Biophysical Research Communications|September 28, 1984
In vivo uptake of insulin into hepatic Golgi fractions: application of the diaminobenzidine-shift protocolD G Kay, M N Khan, B I Posner, et al.
Journal of Virology|January 7, 1998
Transgenic mice expressing human immunodeficiency virus type 1 in immune cells develop a severe AIDS-like diseaseZ Hanna, D G Kay, M Cool, et al.
Cell|October 28, 1998
Nef harbors a major determinant of pathogenicity for an AIDS-like disease induced by HIV-1 in transgenic miceZ Hanna, D G Kay, N Rebai, et al.
Journal of Virology|September 5, 2001
The pathogenicity of human immunodeficiency virus (HIV) type 1 Nef in CD4C/HIV transgenic mice is abolished by mutation of its SH3-binding domain, and disease development is delayed in the absence of HckZ Hanna, X Weng, D G Kay, et al.
Pageof 2

Showing results (1-10 of 17) with videos related to

Sort By:
Pageof 2
Journal of Virology|November 1, 1993
Identification of the infected target cell type in spongiform myeloencephalopathy induced by the neurotropic Cas-Br-E murine leukemia virusC Gravel, D G Kay, P Jolicoeur
Journal of Virology|December 1, 1996
The prion protein gene is dispensable for the development of spongiform myeloencephalopathy induced by the neurovirulent Cas-Br-E murine leukemia virusP Jolicoeur, G Massé, D G Kay
Journal of Bacteriology|March 1, 1980
Ornithine decarboxylase activity and cell cycle regulation in Saccharomyces cerevisiaeD G Kay, R A Singer, G C Johnston
Proceedings of the National Academy of Sciences of the United States of America|February 15, 1991
Retrovirus-induced spongiform myeloencephalopathy in mice: regional distribution of infected target cells and neuronal loss occurring in the absence of viral expression in neuronsD G Kay, C Gravel, Y Robitaille, et al.
Journal of Virology|December 1, 1991
The majority of cells infected with the defective murine AIDS virus belong to the B-cell lineageM Huang, C Simard, D G Kay, et al.
Journal of Virology|August 1, 1990
Substitution of the U3 long terminal repeat region of the neurotropic Cas-Br-E retrovirus affects its disease-inducing potentialY Paquette, D G Kay, E Rassart, et al.
Biochemical and Biophysical Research Communications|September 28, 1984
In vivo uptake of insulin into hepatic Golgi fractions: application of the diaminobenzidine-shift protocolD G Kay, M N Khan, B I Posner, et al.
Journal of Virology|January 7, 1998
Transgenic mice expressing human immunodeficiency virus type 1 in immune cells develop a severe AIDS-like diseaseZ Hanna, D G Kay, M Cool, et al.
Cell|October 28, 1998
Nef harbors a major determinant of pathogenicity for an AIDS-like disease induced by HIV-1 in transgenic miceZ Hanna, D G Kay, N Rebai, et al.
Journal of Virology|September 5, 2001
The pathogenicity of human immunodeficiency virus (HIV) type 1 Nef in CD4C/HIV transgenic mice is abolished by mutation of its SH3-binding domain, and disease development is delayed in the absence of HckZ Hanna, X Weng, D G Kay, et al.
Pageof 2