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Endocrinology|June 1, 1984
Electrolytic lesions of the dorsal rostral pons prevent adrenocorticotropin increases after hemorrhageA M Lefcort, D G Ward, D S GannThe American Journal of Physiology|September 1, 1977
Neurons in medullary areas controlling ACTH: atrial input and rostral projectionsD G Ward, A J Baertschi, D S GannThe American Journal of Physiology|July 1, 1991
Lack of cardiovascular and vasopressin responses to human relaxin in conscious, late-pregnant ratsD G Ward, M J Cronin, A J BaertschiBiochemical and Biophysical Research Communications|July 31, 1992
Relaxin increases rat heart rate by a direct action on the cardiac atriumD G Ward, G R Thomas, M J CroninThe American Journal of Physiology|September 1, 1976
Role of atrial receptors in the control of ACTHA J Baertschi, D G Ward, D S GannPediatric Emergency Care|December 1, 1996
A deep-tissue hemangioma presenting as a rapidly progressive expanding mass and thrombocytopenia in an infantD G Ward, W Wang, W Fesmire, et al.The Journal of Membrane Biology|December 1, 1993
Irreversible effects of calcium ions on the plasma membrane calcium pumpD G Ward, T J Walton, J D CavieresBrain Research|January 6, 1980
Neurons in the dorsal rostral pons process information about changes in venous return and in arterial pressureD G Ward, A M Lefcourt, D S GannEndocrinology|November 1, 1976
Inhibitory and facilitatory areas of the rostral pons mediating ACTH release in the catD G Ward, W E Grizzle, D S GannEndocrinology|April 1, 1978
Inhibitory and facilitatory areas of the ventral midbrain mediating release of corticotropin in the catD G Ward, M G Bolton, D S GannPageof 5