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Arteriosclerosis, Thrombosis, and Vascular Biology|April 9, 1999
Increased atherosclerosis in ApoE and LDL receptor gene knock-out mice as a result of human cholesteryl ester transfer protein transgene expressionA S Plump, L Masucci-Magoulas, C Bruce, et al.
The Journal of Clinical Endocrinology and Metabolism|May 1, 1977
Glycosylated hemoglobins and long-term blood glucose control in diabetes mellitusK H Gabbay, K Hasty, J L Breslow, et al.
The Journal of Clinical Investigation|June 1, 1996
Apolipoprotein A-I is required for cholesteryl ester accumulation in steroidogenic cells and for normal adrenal steroid productionA S Plump, S K Erickson, W Weng, et al.
Journal of Lipid Research|March 21, 1998
ApoA-I deficiency causes both hypertriglyceridemia and increased atherosclerosis in human apoB transgenic miceE Voyiaziakis, I J Goldberg, A S Plump, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 17, 1999
Induced mutant mouse lines that express lipoprotein lipase in cardiac muscle, but not in skeletal muscle and adipose tissue, have normal plasma triglyceride and high-density lipoprotein-cholesterol levelsS Levak-Frank, W Hofmann, P H Weinstock, et al.
The Journal of Clinical Investigation|October 1, 1995
Decreased early atherosclerotic lesions in hypertriglyceridemic mice expressing cholesteryl ester transfer protein transgeneT Hayek, L Masucci-Magoulas, X Jiang, et al.
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