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Gut|April 16, 2005
Octreotide induced prolongation of colonic transit increases faecal anaerobic bacteria, bile acid metabolising enzymes, and serum deoxycholic acid in patients with acromegalyL A Thomas, M J Veysey, G M Murphy, et al.Gut|December 1, 1991
Gastric carcinogenesis in the rat induced by duodenogastric reflux without carcinogens: morphology, mucin histochemistry, polyamine metabolism, and labelling indexP R Taylor, R C Mason, M I Filipe, et al.Occupational Medicine (Oxford, England)|October 5, 2001
Health and safety in the catering industryD GleesonGut|May 1, 1996
Roles of gall bladder emptying and intestinal transit in the pathogenesis of octreotide induced gall bladder stonesS H Hussaini, S P Pereira, M J Veysey, et al.Clinical Science and Molecular Medicine|November 1, 1976
The mechanism for small-bowel adaptation in lactating ratsE Elias, R H DowlingGut|July 1, 1981
Prolactin and the small intestine. Effect of hyperprolactinaemia on mucosal structure in the ratE Muller, R H DowlingGut|April 16, 1999
Prolonged large bowel transit increases serum deoxycholic acid: a risk factor for octreotide induced gallstonesM J Veysey, L A Thomas, A I Mallet, et al.Clinical Science (London, England : 1979)|November 1, 1980
Speed of onset of adaptive mucosal hypoplasia and hypofunction in the intestine of parenterally fed ratsC A Hughes, R H DowlingLancet (London, England)|January 23, 1982
Is recurrence inevitable after gallstone dissolution by bile-acid treatment?D C Ruppin, R H DowlingSchweizerische Medizinische Wochenschrift|July 20, 1985
[Potential adaptation of the gastrointestinal system. Existence of an enteropancreatic trophic axis, the role of hormones and polyamines]R H Dowling, B M MiazzaPageof 52