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D H Christiansen

Showing results (1-10 of 21) with videos related to

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Leukemia|December 26, 2001
Internal tandem duplications of the FLT3 and MLL genes are mainly observed in atypical cases of therapy-related acute myeloid leukemia with a normal karyotype and are unrelated to type of previous therapyD H Christiansen, J Pedersen-Bjergaard
Computer Programs in Biomedicine|September 1, 1981
Guidelines for managing datasets, programs and printouts in scientific researchK E Muller, D H Christiansen, J Smith
Leukemia|September 13, 2003
Methylation of p15INK4B is common, is associated with deletion of genes on chromosome arm 7q and predicts a poor prognosis in therapy-related myelodysplasia and acute myeloid leukemiaD H Christiansen, M K Andersen, J Pedersen-Bjergaard
Blood|May 29, 2000
Therapy-related acute myeloid leukemia and myelodysplasia after high-dose chemotherapy and autologous stem cell transplantationJ Pedersen-Bjergaard, M K Andersen, D H Christiansen
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|March 7, 2001
Mutations with loss of heterozygosity of p53 are common in therapy-related myelodysplasia and acute myeloid leukemia after exposure to alkylating agents and significantly associated with deletion or loss of 5q, a complex karyotype, and a poor prognosisD H Christiansen, M K Andersen, J Pedersen-Bjergaard
Leukemia|December 25, 2004
Amplification or duplication of chromosome band 21q22 with multiple copies of the AML1 gene and mutation of the TP53 gene in therapy-related MDS and AMLM K Andersen, D H Christiansen, J Pedersen-Bjergaard
Leukemia|October 26, 2002
Causality of myelodysplasia and acute myeloid leukemia and their genetic abnormalitiesJ Pedersen-Bjergaard, D H Christiansen, M K Andersen, et al.
Genes, Chromosomes & Cancer|April 3, 2001
Duplication or amplification of chromosome band 11q23, including the unrearranged MLL gene, is a recurrent abnormality in therapy-related MDS and AML, and is closely related to mutation of the TP53 gene and to previous therapy with alkylating agentsM K Andersen, D H Christiansen, M Kirchhoff, et al.
Leukemia|September 23, 2006
Alternative genetic pathways and cooperating genetic abnormalities in the pathogenesis of therapy-related myelodysplasia and acute myeloid leukemiaJ Pedersen-Bjergaard, D H Christiansen, F Desta, et al.
Leukemia|January 21, 2006
Activating mutations of JAK2V617F are uncommon in t-MDS and t-AML and are only observed in atypic casesF Desta, D H Christiansen, M K Andersen, et al.
Pageof 3

Showing results (1-10 of 21) with videos related to

Sort By:
Pageof 3
Leukemia|December 26, 2001
Internal tandem duplications of the FLT3 and MLL genes are mainly observed in atypical cases of therapy-related acute myeloid leukemia with a normal karyotype and are unrelated to type of previous therapyD H Christiansen, J Pedersen-Bjergaard
Computer Programs in Biomedicine|September 1, 1981
Guidelines for managing datasets, programs and printouts in scientific researchK E Muller, D H Christiansen, J Smith
Leukemia|September 13, 2003
Methylation of p15INK4B is common, is associated with deletion of genes on chromosome arm 7q and predicts a poor prognosis in therapy-related myelodysplasia and acute myeloid leukemiaD H Christiansen, M K Andersen, J Pedersen-Bjergaard
Blood|May 29, 2000
Therapy-related acute myeloid leukemia and myelodysplasia after high-dose chemotherapy and autologous stem cell transplantationJ Pedersen-Bjergaard, M K Andersen, D H Christiansen
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|March 7, 2001
Mutations with loss of heterozygosity of p53 are common in therapy-related myelodysplasia and acute myeloid leukemia after exposure to alkylating agents and significantly associated with deletion or loss of 5q, a complex karyotype, and a poor prognosisD H Christiansen, M K Andersen, J Pedersen-Bjergaard
Leukemia|December 25, 2004
Amplification or duplication of chromosome band 21q22 with multiple copies of the AML1 gene and mutation of the TP53 gene in therapy-related MDS and AMLM K Andersen, D H Christiansen, J Pedersen-Bjergaard
Leukemia|October 26, 2002
Causality of myelodysplasia and acute myeloid leukemia and their genetic abnormalitiesJ Pedersen-Bjergaard, D H Christiansen, M K Andersen, et al.
Genes, Chromosomes & Cancer|April 3, 2001
Duplication or amplification of chromosome band 11q23, including the unrearranged MLL gene, is a recurrent abnormality in therapy-related MDS and AML, and is closely related to mutation of the TP53 gene and to previous therapy with alkylating agentsM K Andersen, D H Christiansen, M Kirchhoff, et al.
Leukemia|September 23, 2006
Alternative genetic pathways and cooperating genetic abnormalities in the pathogenesis of therapy-related myelodysplasia and acute myeloid leukemiaJ Pedersen-Bjergaard, D H Christiansen, F Desta, et al.
Leukemia|January 21, 2006
Activating mutations of JAK2V617F are uncommon in t-MDS and t-AML and are only observed in atypic casesF Desta, D H Christiansen, M K Andersen, et al.
Pageof 3