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Physiological Genomics|June 18, 2009
Longitudinal system-based analysis of transcriptional responses to type I interferonsD J Pappas, G Coppola, P A Gabatto, et al.Proceedings of the National Academy of Sciences of the United States of America|July 5, 2001
From hematopoiesis to neuropoiesis: evidence of overlapping genetic programsA V Terskikh, M C Easterday, L Li, et al.Archives of Neurology|October 27, 2001
Association of moderate polyglutamine tract expansions in the slow calcium-activated potassium channel type 3 with ataxiaK P Figueroa, P Chan, L Schöls, et al.Journal of Molecular Neuroscience : MN|January 20, 2018
Analysis of Candidate Genes at the IBGC1 Locus Associated with Idiopathic Basal Ganglia Calcification ("Fahr" Disease')J R M Oliveira, M J Sobrido, E Spiteri, et al.Journal of Molecular Neuroscience : MN|October 6, 2007
Analysis of candidate genes at the IBGC1 locus associated with idiopathic basal ganglia calcification ("Fahr's disease")J R M Oliveira, M J Sobrido, E Spiteri, et al.Neurology|March 12, 2003
Possible association of the tau H1/H1 genotype with primary progressive aphasiaM-J Sobrido, A Abu-Khalil, S Weintraub, et al.Cell Death and Differentiation|March 14, 2009
Molecular stages of rapid and uniform neuralization of human embryonic stem cellsR Bajpai, G Coppola, M Kaul, et al.Molecular Psychiatry|May 21, 2009
High-density SNP association study of the 17q21 chromosomal region linked to autism identifies CACNA1G as a novel candidate geneS P Strom, J L Stone, J R Ten Bosch, et al.Molecular Psychiatry|June 8, 2011
Modeling the functional genomics of autism using human neuronsG Konopka, E Wexler, E Rosen, et al.Annals of Neurology|June 9, 1999
From genotype to phenotype: a clinical pathological, and biochemical investigation of frontotemporal dementia and parkinsonism (FTDP-17) caused by the P301L tau mutationZ S Nasreddine, M Loginov, L N Clark, et al.Pageof 13