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The Journal of Laboratory and Clinical Medicine|October 1, 1993
Endogenous testosterone, fibrinolysis, and coronary heart disease risk in hyperlipidemic menC J Glueck, H I Glueck, D Stroop, et al.
American Journal of Hematology|April 1, 1995
Idiopathic osteonecrosis, hypofibrinolysis, high plasminogen activator inhibitor, high lipoprotein(a), and therapy with StanozololC J Glueck, R Freiberg, H I Glueck, et al.
Thrombosis and Haemostasis|May 3, 1993
Familial high plasminogen activator inhibitor with hypofibrinolysis, a new pathophysiologic cause of osteonecrosis?C J Glueck, H I Glueck, L Mieczkowski, et al.
The American Journal of Clinical Nutrition|June 1, 1982
Changes in plasma lipid and lipoprotein fractions after alteration in dietary cholesterol, polyunsaturated, saturated, and total fat in free-living normal and hypercholesterolemic childrenE A Stein, J Shapero, C McNerney, et al.
The American Journal of the Medical Sciences|May 1, 1994
Beta blockers, Lp(a), hypertension, and reduced basal fibrinolytic activityC J Glueck, H I Glueck, T Hamer, et al.
The Journal of Extra-Corporeal Technology|September 28, 2020
Coagulopathy Characterized by Rotational Thromboelastometry in a Porcine Pediatric ECMO ModelChristopher R Reed, Desiree Bonadonna, Jeffrey Everitt, et al.
The American Journal of Cardiology|January 15, 1995
Evidence that homocysteine is an independent risk factor for atherosclerosis in hyperlipidemic patientsC J Glueck, P Shaw, J E Lang, et al.
Biological Psychiatry|August 15, 1993
Improvement in symptoms of depression and in an index of life stressors accompany treatment of severe hypertriglyceridemiaC J Glueck, M Tieger, R Kunkel, et al.
Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontics|February 25, 1998
A preliminary pilot study of treatment of thrombophilia and hypofibrinolysis and amelioration of the pain of osteonecrosis of the jawsC J Glueck, R E McMahon, J E Bouquot, et al.
The American Journal of Cardiology|September 11, 1999
Effect of exogenous estrogen on atherothrombotic vascular disease risk related to the presence or absence of the factor V Leiden mutation (resistance to activated protein C)C J Glueck, P Wang, R N Fontaine, et al.
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