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D I Graham

Showing results (321-330 of 338) with videos related to

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Acta Neurochirurgica. Supplementum|January 1, 1993
Quantification of primary and secondary lesions in severe head injuryD I Graham, J H Adams, D Doyle, et al.
Neuropathology and Applied Neurobiology|December 13, 2012
The neuroinflammatory response in humans after traumatic brain injuryC Smith, S M Gentleman, P D Leclercq, et al.
Neuropathology and Applied Neurobiology|June 7, 2000
beta-amyloid (Abeta)42(43), abeta42, abeta40 and apoE immunostaining of plaques in fatal head injuryK Horsburgh, G M Cole, F Yang, et al.
Neuroscience|April 6, 2002
Mild traumatic brain injury induces apoptotic cell death in the cortex that is preceded by decreases in cellular Bcl-2 immunoreactivityR Raghupathi, A C Conti, D I Graham, et al.
Clinical Neuropathology|December 5, 2006
No evidence for the presence of apolipoprotein epsilon4, interleukin-lalpha allele 2 and interleukin-1beta allele 2 cause an increase in programmed cell death following traumatic brain injury in humansV E Johnson, L Murray, R Raghupathi, et al.
Annals of Neurology|December 1, 1985
Effect of pretreatment with the calcium antagonist nimodipine on local cerebral blood flow and histopathology after middle cerebral artery occlusionA A Mohamed, O Gotoh, D I Graham, et al.
Neuroreport|April 14, 1997
A beta 42 is the predominant form of amyloid beta-protein in the brains of short-term survivors of head injuryS M Gentleman, B D Greenberg, M J Savage, et al.
Journal of Neurotrauma|May 5, 2001
Pharmacologic inhibition of poly(ADP-ribose) polymerase is neuroprotective following traumatic brain injury in ratsM C LaPlaca, J Zhang, R Raghupathi, et al.
Brain Pathology (Zurich, Switzerland)|February 11, 1998
Glial-neuronal interactions in Alzheimer's disease: the potential role of a 'cytokine cycle' in disease progressionW S Griffin, J G Sheng, M C Royston, et al.
Cellular and Molecular Neurobiology|March 18, 1999
Is there a genetic basis for the deposition of beta-amyloid after fatal head injury?D I Graham, S M Gentleman, J A Nicoll, et al.
Pageof 34

Showing results (321-330 of 338) with videos related to

Sort By:
Pageof 34
Acta Neurochirurgica. Supplementum|January 1, 1993
Quantification of primary and secondary lesions in severe head injuryD I Graham, J H Adams, D Doyle, et al.
Neuropathology and Applied Neurobiology|December 13, 2012
The neuroinflammatory response in humans after traumatic brain injuryC Smith, S M Gentleman, P D Leclercq, et al.
Neuropathology and Applied Neurobiology|June 7, 2000
beta-amyloid (Abeta)42(43), abeta42, abeta40 and apoE immunostaining of plaques in fatal head injuryK Horsburgh, G M Cole, F Yang, et al.
Neuroscience|April 6, 2002
Mild traumatic brain injury induces apoptotic cell death in the cortex that is preceded by decreases in cellular Bcl-2 immunoreactivityR Raghupathi, A C Conti, D I Graham, et al.
Clinical Neuropathology|December 5, 2006
No evidence for the presence of apolipoprotein epsilon4, interleukin-lalpha allele 2 and interleukin-1beta allele 2 cause an increase in programmed cell death following traumatic brain injury in humansV E Johnson, L Murray, R Raghupathi, et al.
Annals of Neurology|December 1, 1985
Effect of pretreatment with the calcium antagonist nimodipine on local cerebral blood flow and histopathology after middle cerebral artery occlusionA A Mohamed, O Gotoh, D I Graham, et al.
Neuroreport|April 14, 1997
A beta 42 is the predominant form of amyloid beta-protein in the brains of short-term survivors of head injuryS M Gentleman, B D Greenberg, M J Savage, et al.
Journal of Neurotrauma|May 5, 2001
Pharmacologic inhibition of poly(ADP-ribose) polymerase is neuroprotective following traumatic brain injury in ratsM C LaPlaca, J Zhang, R Raghupathi, et al.
Brain Pathology (Zurich, Switzerland)|February 11, 1998
Glial-neuronal interactions in Alzheimer's disease: the potential role of a 'cytokine cycle' in disease progressionW S Griffin, J G Sheng, M C Royston, et al.
Cellular and Molecular Neurobiology|March 18, 1999
Is there a genetic basis for the deposition of beta-amyloid after fatal head injury?D I Graham, S M Gentleman, J A Nicoll, et al.
Pageof 34