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Arthritis & Rheumatology (Hoboken, N.J.)|March 29, 2021
Pim Kinases as Therapeutic Targets in Early Rheumatoid ArthritisNicola J Maney, Henrique Lemos, Ben Barron-Millar, et al.Rheumatology (Oxford, England)|August 25, 2023
Differential properties of Janus kinase inhibitors in the treatment of immune-mediated inflammatory diseasesPeter C Taylor, Ernest Choy, Xenofon Baraliakos, et al.Research Involvement and Engagement|October 25, 2017
GRIPP2 reporting checklists: tools to improve reporting of patient and public involvement in researchS Staniszewska, J Brett, I Simera, et al.BMJ (Clinical Research Ed.)|August 4, 2017
GRIPP2 reporting checklists: tools to improve reporting of patient and public involvement in researchS Staniszewska, J Brett, I Simera, et al.Pharmacogenetics and Genomics|March 6, 2009
Investigation of genetic variants within candidate genes of the TNFRSF1B signalling pathway on the response to anti-TNF agents in a UK cohort of rheumatoid arthritis patientsJohn D Bowes, Catherine Potter, Laura J Gibbons, et al.Lancet (London, England)|April 17, 2007
Safety and efficacy of the HIV-1 integrase inhibitor raltegravir (MK-0518) in treatment-experienced patients with multidrug-resistant virus: a phase II randomised controlled trialBeatriz Grinsztejn, Bach-Yen Nguyen, Christine Katlama, et al.Plos One|January 12, 2012
FcγRIIIa expression on monocytes in rheumatoid arthritis: role in immune-complex stimulated TNF production and non-response to methotrexate therapyDawn L Cooper, Stephen G Martin, James I Robinson, et al.Pharmacogenomics|October 30, 2020
Pharmacogenetics of TNF inhibitor response in rheumatoid arthritis utilizing the two-component disease activity scoreSyed Sa Gilani, Nisha Nair, Darren Plant, et al.Annals of the Rheumatic Diseases|May 8, 2010
Association between anti-tumour necrosis factor treatment response and genetic variants within the TLR and NF{kappa}B signalling pathwaysCatherine Potter, Heather J Cordell, Anne Barton, et al.Archives of Disease in Childhood|April 1, 1989
Haemophilus influenzae type b disease in the Oxford regionG Tudor-Williams, J Frankland, D Isaacs, et al.Pageof 79