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Circulation Research|April 1, 1994
Regulation of fos and jun immediate-early genes by redox or metabolic stress in cardiac myocytesK A Webster, D J Discher, N H Bishopric
The Journal of Biological Chemistry|August 5, 1993
Induction and nuclear accumulation of fos and jun proto-oncogenes in hypoxic cardiac myocytesK A Webster, D J Discher, N H Bishopric
Journal of Molecular and Cellular Cardiology|January 1, 1995
Cardioprotection in an in vitro model of hypoxic preconditioningK A Webster, D J Discher, N H Bishopric
Biochemical and Biophysical Research Communications|May 20, 1998
Hypoxia regulates expression of the endothelin-1 gene through a proximal hypoxia-inducible factor-1 binding site on the antisense strandJ Hu, D J Discher, N H Bishopric, et al.
Circulation Research|February 10, 2000
Rapid activation of neutral sphingomyelinase by hypoxia-reoxygenation of cardiac myocytesO M Hernandez, D J Discher, N H Bishopric, et al.
Cardiovascular Research|December 1, 1995
Cell-specificity and signaling pathway of endothelin-1 gene regulation by hypoxiaI Bodi, N H Bishopric, D J Discher, et al.
The Journal of Biological Chemistry|September 25, 1998
Hypoxia regulates beta-enolase and pyruvate kinase-M promoters by modulating Sp1/Sp3 binding to a conserved GC elementD J Discher, N H Bishopric, X Wu, et al.
Molecular and Cellular Biology|March 1, 1996
Physical and functional sensitivity of zinc finger transcription factors to redox changeX Wu, N H Bishopric, D J Discher, et al.
The Journal of Clinical Investigation|August 3, 1999
Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53K A Webster, D J Discher, S Kaiser, et al.
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