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D J Wells

Showing results (51-60 of 58) with videos related to

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FEBS Letters|April 28, 1997
Immune responses, not promoter inactivation, are responsible for decreased long-term expression following plasmid gene transfer into skeletal muscleK E Wells, J Maule, R Kingston, et al.
Experimental Cell Research|August 2, 2005
Mutated fukutin-related protein (FKRP) localises as wild type in differentiated muscle cellsN F Dolatshad, M Brockington, S Torelli, et al.
Human Gene Therapy|December 16, 1998
Insertion of two independent enhancers in the long terminal repeat of a self-inactivating vector results in high-titer retroviral vectors with tissue-specific expressionA Fassati, A Bardoni, M Sironi, et al.
The Journal of Clinical Investigation|August 1, 1997
Genetic correction of dystrophin deficiency and skeletal muscle remodeling in adult MDX mouse via transplantation of retroviral producer cellsA Fassati, D J Wells, P A Sgro Serpente, et al.
Human Molecular Genetics|August 1, 1995
Expression of human full-length and minidystrophin in transgenic mdx mice: implications for gene therapy of Duchenne muscular dystrophyD J Wells, K E Wells, E A Asante, et al.
Human Molecular Genetics|April 1, 1992
Human dystrophin expression corrects the myopathic phenotype in transgenic mdx miceD J Wells, K E Wells, F S Walsh, et al.
Bone|June 23, 2004
Bone biomechanical properties in LRP5 mutant miceM P Akhter, D J Wells, S J Short, et al.
Laboratory Animals|April 8, 2006
Assessing the welfare of genetically altered miceD J Wells, L C Playle, W E J Enser, et al.
Pageof 6

Showing results (51-60 of 58) with videos related to

Sort By:
Pageof 6
You have reached the last page of results.This site can display upto 58 results.
FEBS Letters|April 28, 1997
Immune responses, not promoter inactivation, are responsible for decreased long-term expression following plasmid gene transfer into skeletal muscleK E Wells, J Maule, R Kingston, et al.
Experimental Cell Research|August 2, 2005
Mutated fukutin-related protein (FKRP) localises as wild type in differentiated muscle cellsN F Dolatshad, M Brockington, S Torelli, et al.
Human Gene Therapy|December 16, 1998
Insertion of two independent enhancers in the long terminal repeat of a self-inactivating vector results in high-titer retroviral vectors with tissue-specific expressionA Fassati, A Bardoni, M Sironi, et al.
The Journal of Clinical Investigation|August 1, 1997
Genetic correction of dystrophin deficiency and skeletal muscle remodeling in adult MDX mouse via transplantation of retroviral producer cellsA Fassati, D J Wells, P A Sgro Serpente, et al.
Human Molecular Genetics|August 1, 1995
Expression of human full-length and minidystrophin in transgenic mdx mice: implications for gene therapy of Duchenne muscular dystrophyD J Wells, K E Wells, E A Asante, et al.
Human Molecular Genetics|April 1, 1992
Human dystrophin expression corrects the myopathic phenotype in transgenic mdx miceD J Wells, K E Wells, F S Walsh, et al.
Bone|June 23, 2004
Bone biomechanical properties in LRP5 mutant miceM P Akhter, D J Wells, S J Short, et al.
Laboratory Animals|April 8, 2006
Assessing the welfare of genetically altered miceD J Wells, L C Playle, W E J Enser, et al.
Pageof 6