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Neuroscience|January 25, 2011
Laminar-specific and developmental expression of aquaporin-4 in the mouse hippocampusM S Hsu, M Seldin, D J Lee, et al.
ASN Neuro|October 16, 2025
Combined Treatment with Minocycline and an mGluR5 Antagonist Alters Resting EEG Spectral Power, but Not Sound-Evoked Responses, in a Mouse Model of Fragile X SyndromeM H Kassir, J W Lovelace, D K Binder, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 10, 1999
Selective inhibition of kindling development by intraventricular administration of TrkB receptor bodyD K Binder, M J Routbort, T E Ryan, et al.
Neuroscience|November 25, 2004
Molecular mechanisms of brain tumor edemaM C Papadopoulos, S Saadoun, D K Binder, et al.
Acta Neurochirurgica. Supplement|February 3, 2004
Aquaporin-1 expression in human glial tumors suggests a potential novel therapeutic target for tumor-associated edemaK Oshio, D K Binder, A Bollen, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 15, 1996
Null mutation of c-fos impairs structural and functional plasticities in the kindling model of epilepsyY Watanabe, R S Johnson, L S Butler, et al.
Biorxiv : the Preprint Server for Biology|January 13, 2025
Enhanced CB1 receptor function in GABAergic neurons mediates hyperexcitability and impaired sensory-driven synchrony of cortical circuits in Fragile X Syndrome model miceD Gonzalez, C R Jonak, M Bernabucci, et al.
Molecular Psychiatry|November 22, 2025
Enhanced CB1 receptor function in GABAergic neurons mediates hyperexcitability and impaired sensory-driven synchrony of cortical circuits in Fragile X Syndrome model miceD Gonzalez, C R Jonak, M Bernabucci, et al.
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