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Journal of Molecular Medicine (Berlin, Germany)|November 24, 1998
Physiological consequences of beta-adrenergic receptor disruptionD K Rohrer
Physiological Reviews|February 11, 1998
G protein-coupled receptors: functional and mechanistic insights through altered gene expressionD K Rohrer, B K Kobilka
Somatic Cell and Molecular Genetics|September 1, 1996
Defective myogenesis in NFB-s mutant associated with a saturable suppression of MYF5 activityD K Rohrer, H M Blau
Annual Review of Pharmacology and Toxicology|May 23, 1998
Insights from in vivo modification of adrenergic receptor gene expressionD K Rohrer, B K Kobilka
The Journal of Biological Chemistry|May 5, 1991
Influence of thyroid hormone and retinoic acid on slow sarcoplasmic reticulum Ca2+ ATPase and myosin heavy chain alpha gene expression in cardiac myocytes. Delineation of cis-active DNA elements that confer responsiveness to thyroid hormone but not to retinoic acidD K Rohrer, R Hartong, W H Dillmann
Molecular and Cellular Endocrinology|September 1, 1992
Thyroid hormone response of slow and fast sarcoplasmic reticulum Ca2+ ATPase mRNA in striated muscleM R Sayen, D K Rohrer, W H Dillmann
The Journal of Biological Chemistry|June 8, 1999
Cardiovascular and metabolic alterations in mice lacking both beta1- and beta2-adrenergic receptorsD K Rohrer, A Chruscinski, E H Schauble, et al.
The American Journal of Physiology|May 12, 1998
Alterations in dynamic heart rate control in the beta 1-adrenergic receptor knockout mouseD K Rohrer, E H Schauble, K H Desai, et al.
The Journal of Biological Chemistry|June 8, 1999
Targeted disruption of the beta2 adrenergic receptor geneA J Chruscinski, D K Rohrer, E Schauble, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 9, 1996
Targeted disruption of the mouse beta1-adrenergic receptor gene: developmental and cardiovascular effectsD K Rohrer, K H Desai, J R Jasper, et al.
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