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Leukemia Research|October 31, 2001
Quantitative, real-time polymerase chain reactions for FLT3 internal tandem duplications are highly sensitive and specificD L Stirewalt, C L Willman, J P RadichBlood Cancer Journal|May 3, 2014
Copy-neutral loss of heterozygosity is prevalent and a late event in the pathogenesis of FLT3/ITD AMLD L Stirewalt, E L Pogosova-Agadjanyan, K Tsuchiya, et al.Leukemia|July 10, 1999
p73 mutations and expression in adult de novo acute myelogenous leukemiaD L Stirewalt, B Clurman, F R Appelbaum, et al.Leukemia Research|January 16, 2003
Mevastatin can increase toxicity in primary AMLs exposed to standard therapeutic agents, but statin efficacy is not simply associated with ras hotspot mutations or overexpressionD L Stirewalt, F R Appelbaum, C L Willman, et al.Blood|May 23, 2001
FLT3, RAS, and TP53 mutations in elderly patients with acute myeloid leukemiaD L Stirewalt, K J Kopecky, S Meshinchi, et al.Blood|January 3, 2001
Prevalence and prognostic significance of Flt3 internal tandem duplication in pediatric acute myeloid leukemiaS Meshinchi, W G Woods, D L Stirewalt, et al.Leukemia|September 3, 2004
A distinctive nuclear morphology in acute myeloid leukemia is strongly associated with loss of HLA-DR expression and FLT3 internal tandem duplicationS J Kussick, D L Stirewalt, H S Yi, et al.Leukemia|May 31, 2017
Mitoxantrone, etoposide and cytarabine following epigenetic priming with decitabine in adults with relapsed/refractory acute myeloid leukemia or other high-grade myeloid neoplasms: a phase 1/2 studyA B Halpern, M Othus, E M Huebner, et al.Leukemia|April 9, 2010
Molecular alterations of the IDH1 gene in AML: a Children's Oncology Group and Southwest Oncology Group studyP A Ho, T A Alonzo, K J Kopecky, et al.Pageof 1