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American Journal of Obstetrics and Gynecology|March 21, 1998
Low risk of perinatal transmission of human papillomavirus: results from a prospective cohort studyD H Watts, L A Koutsky, K K Holmes, et al.Journal of Immunology (Baltimore, Md. : 1950)|April 15, 1992
Induction of cytotoxic T lymphocytes specific for a syngeneic tumor expressing the E6 oncoprotein of human papillomavirus type 16L Chen, M T Mizuno, M C Singhal, et al.The Journal of Infectious Diseases|April 8, 2000
Detection of cervical antibodies to human papillomavirus type 16 (HPV-16) capsid antigens in relation to detection of HPV-16 DNA and cervical lesionsM E Hagensee, L A Koutsky, S K Lee, et al.Journal of Virology|March 1, 1991
Characterization of murine polyclonal antisera and monoclonal antibodies generated against intact and denatured human papillomavirus type 1 virionsN Yaegashi, S A Jenison, J M Valentine, et al.Genome Research|January 24, 1998
The complete set of predicted genes from Saccharomyces cerevisiae in a readily usable formJ R Hudson, E P Dawson, K L Rushing, et al.The Journal of Infectious Diseases|June 6, 2000
Comparison of human papillomavirus types 16, 18, and 6 capsid antibody responses following incident infectionJ J Carter, L A Koutsky, J P Hughes, et al.Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|October 28, 1997
Inactivation of G2 checkpoint function and chromosomal destabilization are linked in human fibroblasts expressing human papillomavirus type 16 E6W K Kaufmann, J L Schwartz, J C Hurt, et al.Cancer Research|December 4, 2001
Serologic evidence of herpes simplex virus 1 infection and oropharyngeal cancer riskJ R Starr, J R Daling, E D Fitzgibbons, et al.Cancer Epidemiology, Biomarkers & Prevention : a Publication of the American Association for Cancer Research, Cosponsored by the American Society of Preventive Oncology|May 1, 1992
The association of human papillomavirus antibodies with cervical cancer riskM T Mandelson, S A Jenison, K J Sherman, et al.Nature Medicine|January 1, 1996
Loss of normal p53 function confers sensitization to Taxol by increasing G2/M arrest and apoptosisA F Wahl, K L Donaldson, C Fairchild, et al.Pageof 12