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Science (New York, N.Y.)|February 2, 1996
IRS-1-mediated inhibition of insulin receptor tyrosine kinase activity in TNF-alpha- and obesity-induced insulin resistanceG S Hotamisligil, P Peraldi, A Budavari, et al.
The Journal of Biological Chemistry|September 21, 2000
Contrasting effects of IRS-1 versus IRS-2 gene disruption on carbohydrate and lipid metabolism in vivoS F Previs, D J Withers, J M Ren, et al.
Pharmacology, Biochemistry, and Behavior|June 1, 1994
Mouse strain differences in the behavioral effects of corticotropin-releasing factor (CRF) and the CRF antagonist alpha-helical CRF9-41L H Conti, D G Costello, L A Martin, et al.
Archives of Otolaryngology (Chicago, Ill. : 1960)|November 1, 1976
Conservative face lift surgeryR C Webster, T M Davidson, M F White, et al.
Biochemical and Biophysical Research Communications|October 16, 1989
Binding kinetics of mutated insulin receptors in transfected cells grown in suspension culture: application to the Tyr----Phe 960 insulin receptor mutantR M Shymko, N S Gonzales, J M Backer, et al.
Biochemistry|December 10, 1996
Ca2+ regulates calmodulin binding to IQ motifs in IRS-1H G Munshi, D J Burks, J L Joyal, et al.
The Journal of Biological Chemistry|June 10, 1985
Predominance of tyrosine phosphorylation of insulin receptors during the initial response of intact cells to insulinD T Pang, B R Sharma, J A Shafer, et al.
The Journal of Biological Chemistry|October 3, 1998
The COOH-terminal tyrosine phosphorylation sites on IRS-1 bind SHP-2 and negatively regulate insulin signalingM G Myers, R Mendez, P Shi, et al.
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