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The American Journal of Physiology|March 1, 1989
Epithelial polarity following ischemia: a requirement for normal cell functionD M Spiegel, P D Wilson, B A MolitorisKidney International|September 1, 1990
Mild ischemia predisposes the S3 segment to gentamicin toxicityD M Spiegel, P F Shanley, B A MolitorisThe Journal of Clinical Investigation|December 1, 1985
Ischemia induces partial loss of surface membrane polarity and accumulation of putative calcium ionophoresB A Molitoris, P D Wilson, R W Schrier, et al.The Journal of Membrane Biology|December 1, 1988
Characterization of ischemia-induced loss of epithelial polarityB A Molitoris, C A Hoilien, R Dahl, et al.The American Journal of Physiology|April 1, 1997
Putting the actin cytoskeleton into perspective: pathophysiology of ischemic alterationsB A MolitorisThe American Journal of Physiology|November 1, 1993
Na(+)-K(+)-ATPase that redistributes to apical membrane during ATP depletion remains functionalB A MolitorisThe American Journal of Physiology|June 1, 1991
Ischemia-induced loss of epithelial polarity: potential role of the actin cytoskeletonB A MolitorisCurrent Opinion in Nephrology and Hypertension|July 1, 1997
Cell biology of aminoglycoside nephrotoxicity: newer aspectsB A MolitorisJournal of the American Society of Nephrology : JASN|June 1, 1991
New insights into the cell biology of ischemic acute renal failureB A MolitorisPageof 32