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Biochemical and Biophysical Research Communications
|
August 31, 1987
Antisense RNA to the c-fos gene: restoration of density-dependent growth arrest in a transformed cell line
D Mercola, A Rundell, J Westwick, et al.
Cancer Gene Therapy
|
February 26, 1998
Suppression of growth and transformation and induction of apoptosis by EGR-1
C Liu, V M Rangnekar, E Adamson, et al.
The Journal of Biological Chemistry
|
May 16, 2001
Protective role for c-Jun in the cellular response to DNA damage
O Potapova, S Basu, D Mercola, et al.
Oncogene
|
September 1, 1992
Rapid, complete and reversible transformation by v-sis precedes irreversible transformation
D Mercola, P M Carpenter, A Grover-Bardwick, et al.
Nature
|
May 13, 1976
Structure of insulin in 4-zinc insulin
G Bentley, E Dodson, G Dodson, et al.
Critical Reviews in Oncogenesis
|
January 1, 1996
EGR-1, the reluctant suppression factor: EGR-1 is known to function in the regulation of growth, differentiation, and also has significant tumor suppressor activity and a mechanism involving the induction of TGF-beta1 is postulated to account for this suppressor activity
C Liu, A Calogero, G Ragona, et al.
Cell Death and Differentiation
|
September 23, 2006
A network of p73, p53 and Egr1 is required for efficient apoptosis in tumor cells
J Yu, V Baron, D Mercola, et al.
Methods in Enzymology
|
November 24, 1999
Antisense methods for discrimination of phenotypic properties of closely related gene products: Jun kinase family
F Bost, R McKay, N M Dean, et al.
Cancer Gene Therapy
|
September 3, 2005
The transcription factor Egr1 is a direct regulator of multiple tumor suppressors including TGFbeta1, PTEN, p53, and fibronectin
V Baron, E D Adamson, A Calogero, et al.
Oncogene
|
May 1, 1994
Suppression of v-sis-dependent transformation by the transcription factor, Egr-1
R P Huang, T Darland, D Okamura, et al.
Page
of 6
Search research articles
Search
Showing results (21-30 of 58) with videos related to
Sort By:
Page
of 6
Biochemical and Biophysical Research Communications
|
August 31, 1987
Antisense RNA to the c-fos gene: restoration of density-dependent growth arrest in a transformed cell line
D Mercola, A Rundell, J Westwick, et al.
Cancer Gene Therapy
|
February 26, 1998
Suppression of growth and transformation and induction of apoptosis by EGR-1
C Liu, V M Rangnekar, E Adamson, et al.
The Journal of Biological Chemistry
|
May 16, 2001
Protective role for c-Jun in the cellular response to DNA damage
O Potapova, S Basu, D Mercola, et al.
Oncogene
|
September 1, 1992
Rapid, complete and reversible transformation by v-sis precedes irreversible transformation
D Mercola, P M Carpenter, A Grover-Bardwick, et al.
Nature
|
May 13, 1976
Structure of insulin in 4-zinc insulin
G Bentley, E Dodson, G Dodson, et al.
Critical Reviews in Oncogenesis
|
January 1, 1996
EGR-1, the reluctant suppression factor: EGR-1 is known to function in the regulation of growth, differentiation, and also has significant tumor suppressor activity and a mechanism involving the induction of TGF-beta1 is postulated to account for this suppressor activity
C Liu, A Calogero, G Ragona, et al.
Cell Death and Differentiation
|
September 23, 2006
A network of p73, p53 and Egr1 is required for efficient apoptosis in tumor cells
J Yu, V Baron, D Mercola, et al.
Methods in Enzymology
|
November 24, 1999
Antisense methods for discrimination of phenotypic properties of closely related gene products: Jun kinase family
F Bost, R McKay, N M Dean, et al.
Cancer Gene Therapy
|
September 3, 2005
The transcription factor Egr1 is a direct regulator of multiple tumor suppressors including TGFbeta1, PTEN, p53, and fibronectin
V Baron, E D Adamson, A Calogero, et al.
Oncogene
|
May 1, 1994
Suppression of v-sis-dependent transformation by the transcription factor, Egr-1
R P Huang, T Darland, D Okamura, et al.
Page
of 6