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Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research
|
September 2, 1999
Inhibition of the Jun kinase pathway blocks DNA repair, enhances p53-mediated apoptosis and promotes gene amplification
R A Gjerset, S Lebedeva, A Haghighi, et al.
Journal of Cellular Biochemistry
|
September 15, 1997
Reciprocal modulation between Sp1 and Egr-1
R P Huang, Y Fan, Z Ni, et al.
Cancer Research
|
November 1, 1995
Egr-1 negatively regulates human tumor cell growth via the DNA-binding domain
R P Huang, C Liu, Y Fan, et al.
Biochimica Et Biophysica Acta
|
July 21, 1978
Comparison of the calcium- and magnesium-induced structural changes of troponin--C. A proton magnetic resonance study
B A Levine, J M Thornton, R Fernandes, et al.
Molecular and Cellular Biology
|
February 18, 1999
The Jun kinase 2 isoform is preferentially required for epidermal growth factor-induced transformation of human A549 lung carcinoma cells
F Bost, R McKay, M Bost, et al.
The Journal of Biological Chemistry
|
October 9, 1999
Differential effect of retinoic acid on growth regulation by phorbol ester in human cancer cell lines
A Agadir, G q Chen, F Bost, et al.
Journal of Theoretical Biology
|
February 7, 1979
Calcium binding by troponin-C and homologs is correlated with the position and linear density of "beta-turn forming" residues
H P Vogt, W Strassburger, A Wollmer, et al.
Gene
|
December 10, 1988
Analysis of a transformed cell line using antisense c-fos RNA
D Mercola, J Westwick, A Y Rundell, et al.
The Journal of Biological Chemistry
|
February 6, 1999
The transcription factor EGR-1 suppresses transformation of human fibrosarcoma HT1080 cells by coordinated induction of transforming growth factor-beta1, fibronectin, and plasminogen activator inhibitor-1
C Liu, J Yao, I de Belle, et al.
The Journal of Biological Chemistry
|
May 30, 1997
The Jun kinase/stress-activated protein kinase pathway functions to regulate DNA repair and inhibition of the pathway sensitizes tumor cells to cisplatin
O Potapova, A Haghighi, F Bost, et al.
Page
of 6
Search research articles
Search
Showing results (31-40 of 58) with videos related to
Sort By:
Page
of 6
Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research
|
September 2, 1999
Inhibition of the Jun kinase pathway blocks DNA repair, enhances p53-mediated apoptosis and promotes gene amplification
R A Gjerset, S Lebedeva, A Haghighi, et al.
Journal of Cellular Biochemistry
|
September 15, 1997
Reciprocal modulation between Sp1 and Egr-1
R P Huang, Y Fan, Z Ni, et al.
Cancer Research
|
November 1, 1995
Egr-1 negatively regulates human tumor cell growth via the DNA-binding domain
R P Huang, C Liu, Y Fan, et al.
Biochimica Et Biophysica Acta
|
July 21, 1978
Comparison of the calcium- and magnesium-induced structural changes of troponin--C. A proton magnetic resonance study
B A Levine, J M Thornton, R Fernandes, et al.
Molecular and Cellular Biology
|
February 18, 1999
The Jun kinase 2 isoform is preferentially required for epidermal growth factor-induced transformation of human A549 lung carcinoma cells
F Bost, R McKay, M Bost, et al.
The Journal of Biological Chemistry
|
October 9, 1999
Differential effect of retinoic acid on growth regulation by phorbol ester in human cancer cell lines
A Agadir, G q Chen, F Bost, et al.
Journal of Theoretical Biology
|
February 7, 1979
Calcium binding by troponin-C and homologs is correlated with the position and linear density of "beta-turn forming" residues
H P Vogt, W Strassburger, A Wollmer, et al.
Gene
|
December 10, 1988
Analysis of a transformed cell line using antisense c-fos RNA
D Mercola, J Westwick, A Y Rundell, et al.
The Journal of Biological Chemistry
|
February 6, 1999
The transcription factor EGR-1 suppresses transformation of human fibrosarcoma HT1080 cells by coordinated induction of transforming growth factor-beta1, fibronectin, and plasminogen activator inhibitor-1
C Liu, J Yao, I de Belle, et al.
The Journal of Biological Chemistry
|
May 30, 1997
The Jun kinase/stress-activated protein kinase pathway functions to regulate DNA repair and inhibition of the pathway sensitizes tumor cells to cisplatin
O Potapova, A Haghighi, F Bost, et al.
Page
of 6