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D Milner

Showing results (621-630 of 658) with videos related to

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JCI Insight|October 26, 2016
Extrapulmonary <i>Aspergillus</i> infection in patients with CARD9 deficiencyNikolaus Rieber, Roel P Gazendam, Alexandra F Freeman, et al.
Annals of the Rheumatic Diseases|February 23, 2021
Systematic evaluation of nine monogenic autoinflammatory diseases reveals common and disease-specific correlations with allergy-associated featuresDaniella Muallem Schwartz, Moses M Kitakule, Brian Lp Dizon, et al.
American Journal of Human Genetics|November 17, 2020
Multiplexed Functional Assessment of Genetic Variants in CARD11Iana Meitlis, Eric J Allenspach, Bradly M Bauman, et al.
Blood|April 27, 2016
Heterozygous STAT1 gain-of-function mutations underlie an unexpectedly broad clinical phenotypeJulie Toubiana, Satoshi Okada, Julia Hiller, et al.
Cell|December 22, 2023
Sensory neurons promote immune homeostasis in the lungMasato Tamari, Kate L Del Bel, Aaron M Ver Heul, et al.
Nature Genetics|November 8, 2016
Elevated basal serum tryptase identifies a multisystem disorder associated with increased TPSAB1 copy numberJonathan J Lyons, Xiaomin Yu, Jason D Hughes, et al.
The Journal of Clinical Investigation|March 1, 2021
Epicutaneous Staphylococcus aureus induces IL-36 to enhance IgE production and ensuing allergic diseaseGarrett J Patrick, Haiyun Liu, Martin P Alphonse, et al.
The Journal of Allergy and Clinical Immunology|April 2, 2013
Signal transducer and activator of transcription 1 (STAT1) gain-of-function mutations and disseminated coccidioidomycosis and histoplasmosisElizabeth P Sampaio, Amy P Hsu, Joseph Pechacek, et al.
The Journal of Experimental Medicine|April 2, 2024
Individuals with JAK1 variants are affected by syndromic features encompassing autoimmunity, atopy, colitis, and dermatitisMichael E Horesh, Marta Martin-Fernandez, Conor Gruber, et al.
The Journal of Experimental Medicine|June 26, 2019
Loss of the interleukin-6 receptor causes immunodeficiency, atopy, and abnormal inflammatory responsesSarah Spencer, Sevgi Köstel Bal, William Egner, et al.
Pageof 66

Showing results (621-630 of 658) with videos related to

Sort By:
Pageof 66
JCI Insight|October 26, 2016
Extrapulmonary <i>Aspergillus</i> infection in patients with CARD9 deficiencyNikolaus Rieber, Roel P Gazendam, Alexandra F Freeman, et al.
Annals of the Rheumatic Diseases|February 23, 2021
Systematic evaluation of nine monogenic autoinflammatory diseases reveals common and disease-specific correlations with allergy-associated featuresDaniella Muallem Schwartz, Moses M Kitakule, Brian Lp Dizon, et al.
American Journal of Human Genetics|November 17, 2020
Multiplexed Functional Assessment of Genetic Variants in CARD11Iana Meitlis, Eric J Allenspach, Bradly M Bauman, et al.
Blood|April 27, 2016
Heterozygous STAT1 gain-of-function mutations underlie an unexpectedly broad clinical phenotypeJulie Toubiana, Satoshi Okada, Julia Hiller, et al.
Cell|December 22, 2023
Sensory neurons promote immune homeostasis in the lungMasato Tamari, Kate L Del Bel, Aaron M Ver Heul, et al.
Nature Genetics|November 8, 2016
Elevated basal serum tryptase identifies a multisystem disorder associated with increased TPSAB1 copy numberJonathan J Lyons, Xiaomin Yu, Jason D Hughes, et al.
The Journal of Clinical Investigation|March 1, 2021
Epicutaneous Staphylococcus aureus induces IL-36 to enhance IgE production and ensuing allergic diseaseGarrett J Patrick, Haiyun Liu, Martin P Alphonse, et al.
The Journal of Allergy and Clinical Immunology|April 2, 2013
Signal transducer and activator of transcription 1 (STAT1) gain-of-function mutations and disseminated coccidioidomycosis and histoplasmosisElizabeth P Sampaio, Amy P Hsu, Joseph Pechacek, et al.
The Journal of Experimental Medicine|April 2, 2024
Individuals with JAK1 variants are affected by syndromic features encompassing autoimmunity, atopy, colitis, and dermatitisMichael E Horesh, Marta Martin-Fernandez, Conor Gruber, et al.
The Journal of Experimental Medicine|June 26, 2019
Loss of the interleukin-6 receptor causes immunodeficiency, atopy, and abnormal inflammatory responsesSarah Spencer, Sevgi Köstel Bal, William Egner, et al.
Pageof 66