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Acta Neuropathologica Communications|June 23, 2022
Pathological and neurophysiological outcomes of seeding human-derived tau pathology in the APP-KI NL-G-F and NL-NL mouse models of Alzheimer's DiseaseS Tok, H Maurin, C Delay, et al.
Biochimica Et Biophysica Acta|January 19, 1995
Amyloid precursor protein is not processed by furin, PACE 4, PC1/3, PC2, PC4 and PC5/6 of the furin family of proprotein processing enzymesB De Strooper, J W Creemers, D Moechars, et al.
Scientific Reports|May 13, 2022
Neurophysiological effects of human-derived pathological tau conformers in the APPKM670/671NL.PS1/L166P amyloid mouse model of Alzheimer's diseaseS Tok, H Maurin, C Delay, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 31, 2000
Aging increased amyloid peptide and caused amyloid plaques in brain of old APP/V717I transgenic mice by a different mechanism than mutant presenilin1I Dewachter, J Van Dorpe, L Smeijers, et al.
The Journal of Biological Chemistry|February 7, 1997
Phosphorylation, subcellular localization, and membrane orientation of the Alzheimer's disease-associated presenilinsB De Strooper, M Beullens, B Contreras, et al.
The Journal of Biological Chemistry|March 30, 2001
Mutant presenilins disturb neuronal calcium homeostasis in the brain of transgenic mice, decreasing the threshold for excitotoxicity and facilitating long-term potentiationI Schneider, D Reverse, I Dewachter, et al.
The Journal of Biological Chemistry|February 26, 1999
Early phenotypic changes in transgenic mice that overexpress different mutants of amyloid precursor protein in brainD Moechars, I Dewachter, K Lorent, et al.
Scientific Reports|October 29, 2017
Emergence of early alterations in network oscillations and functional connectivity in a tau seeding mouse model of Alzheimer's disease pathologyA Ahnaou, D Moechars, L Raeymaekers, et al.
Neurobiology of Disease|February 15, 2000
Behavioral disturbances without amyloid deposits in mice overexpressing human amyloid precursor protein with Flemish (A692G) or Dutch (E693Q) mutationS Kumar-Singh, I Dewachter, D Moechars, et al.
Cell Death and Differentiation|November 15, 2014
GSK-3β dysregulation contributes to parkinson's-like pathophysiology with associated region-specific phosphorylation and accumulation of tau and α-synucleinJ J Credle, J L George, J Wills, et al.
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