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The American Journal of Pathology|December 1, 1988
The presence of heparan sulfate proteoglycans in the neuritic plaques and congophilic angiopathy in Alzheimer's diseaseA D Snow, H Mar, D Nochlin, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 13, 1999
Missense and silent tau gene mutations cause frontotemporal dementia with parkinsonism-chromosome 17 type, by affecting multiple alternative RNA splicing regulatory elementsI D'Souza, P Poorkaj, M Hong, et al.
Neurology|March 1, 1997
Clinical-neuropathologic findings in multi-infarct dementia: a report of six autopsied casesC Hulette, D Nochlin, D McKeel, et al.
The American Journal of Pathology|December 10, 1998
Transgenic mice over-expressing the C-99 fragment of betaPP with an alpha-secretase site mutation develop a myopathy similar to human inclusion body myositisL W Jin, M G Hearn, C E Ogburn, et al.
Journal of Neuropathology and Experimental Neurology|January 5, 2002
Hyperphosphorylation of RNA polymerase II and reduced neuronal RNA levels precede neurofibrillary tangles in Alzheimer diseaseJ W Husseman, J L Hallows, D B Bregman, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|November 1, 1990
Immunolocalization of heparan sulfate proteoglycans to the prion protein amyloid plaques of Gerstmann-Straussler syndrome, Creutzfeldt-Jakob disease and scrapieA D Snow, T N Wight, D Nochlin, et al.
Progress in Clinical and Biological Research|January 1, 1989
Characteristics of familial Alzheimer's disease in nine kindreds of Volga German ancestryT D Bird, T H Lampe, E J Nemens, et al.
American Journal of Human Genetics|January 13, 2000
The number of trait loci in late-onset Alzheimer diseaseE W Daw, H Payami, E J Nemens, et al.
Neurology|July 1, 1989
Familial dementia with PrP-positive amyloid plaques: a variant of Gerstmann-Sträussler syndromeD Nochlin, S M Sumi, T D Bird, et al.
Neuron|January 1, 1994
An important role of heparan sulfate proteoglycan (Perlecan) in a model system for the deposition and persistence of fibrillar A beta-amyloid in rat brainA D Snow, R Sekiguchi, D Nochlin, et al.
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