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Showing results (301-310 of 380) with videos related to

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Journal of the American Society of Nephrology : JASN|February 23, 2007
Inhibition of p38 mitogen-activated protein kinase is effective in the treatment of experimental crescentic glomerulonephritis and suppresses monocyte chemoattractant protein-1 but not IL-1beta or IL-6Abdulmunem Sheryanna, Gurjeet Bhangal, John McDaid, et al.
Nephron. Experimental Nephrology|September 1, 2005
Interleukin-11 reduces renal injury and glomerular NF-kappa B activity in murine experimental glomerulonephritisP C Lai, J Smith, G Bhangal, et al.
The Journal of Clinical Investigation|April 22, 2010
Mannose receptor interacts with Fc receptors and is critical for the development of crescentic glomerulonephritis in miceKonstantia-Maria Chavele, Luisa Martinez-Pomares, Jan Domin, et al.
Kidney International|April 20, 2020
Spleen tyrosine kinase inhibition is an effective treatment for established vasculitis in a pre-clinical modelStephen P McAdoo, Maria Prendecki, Anisha Tanna, et al.
Kidney International|June 26, 2002
Fragmentation of filtered proteins and implications for glomerular protein sieving in Fanconi syndromeAnthony G W Norden, Marta Lapsley, Philip J Lee, et al.
The Journal of Pathology|June 14, 2021
Characterisation of an enhanced preclinical model of experimental MPO-ANCA autoimmune vasculitisMaria Prendecki, Kavita Gulati, Tabitha Turner-Stokes, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 4, 2012
FcγRIIb on myeloid cells and intrinsic renal cells rather than B cells protects from nephrotoxic nephritisPhoebe E H Sharp, Javier Martin-Ramirez, Sara M Mangsbo, et al.
Molecular Immunology|January 17, 2012
Increased incidence of anti-GBM disease in Fcgamma receptor 2b deficient mice, but not mice with conditional deletion of Fcgr2b on either B cells or myeloid cells alonePhoebe E H Sharp, Javier Martin-Ramirez, Peter Boross, et al.
Kidney International|July 5, 2017
Circulating complement factor H-related proteins 1 and 5 correlate with disease activity in IgA nephropathyNicholas R Medjeral-Thomas, Hannah J Lomax-Browne, Hannah Beckwith, et al.
Kidney International|May 17, 2017
Patients double-seropositive for ANCA and anti-GBM antibodies have varied renal survival, frequency of relapse, and outcomes compared to single-seropositive patientsStephen P McAdoo, Anisha Tanna, Zdenka Hrušková, et al.
Pageof 38

Showing results (301-310 of 380) with videos related to

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Pageof 38
Journal of the American Society of Nephrology : JASN|February 23, 2007
Inhibition of p38 mitogen-activated protein kinase is effective in the treatment of experimental crescentic glomerulonephritis and suppresses monocyte chemoattractant protein-1 but not IL-1beta or IL-6Abdulmunem Sheryanna, Gurjeet Bhangal, John McDaid, et al.
Nephron. Experimental Nephrology|September 1, 2005
Interleukin-11 reduces renal injury and glomerular NF-kappa B activity in murine experimental glomerulonephritisP C Lai, J Smith, G Bhangal, et al.
The Journal of Clinical Investigation|April 22, 2010
Mannose receptor interacts with Fc receptors and is critical for the development of crescentic glomerulonephritis in miceKonstantia-Maria Chavele, Luisa Martinez-Pomares, Jan Domin, et al.
Kidney International|April 20, 2020
Spleen tyrosine kinase inhibition is an effective treatment for established vasculitis in a pre-clinical modelStephen P McAdoo, Maria Prendecki, Anisha Tanna, et al.
Kidney International|June 26, 2002
Fragmentation of filtered proteins and implications for glomerular protein sieving in Fanconi syndromeAnthony G W Norden, Marta Lapsley, Philip J Lee, et al.
The Journal of Pathology|June 14, 2021
Characterisation of an enhanced preclinical model of experimental MPO-ANCA autoimmune vasculitisMaria Prendecki, Kavita Gulati, Tabitha Turner-Stokes, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 4, 2012
FcγRIIb on myeloid cells and intrinsic renal cells rather than B cells protects from nephrotoxic nephritisPhoebe E H Sharp, Javier Martin-Ramirez, Sara M Mangsbo, et al.
Molecular Immunology|January 17, 2012
Increased incidence of anti-GBM disease in Fcgamma receptor 2b deficient mice, but not mice with conditional deletion of Fcgr2b on either B cells or myeloid cells alonePhoebe E H Sharp, Javier Martin-Ramirez, Peter Boross, et al.
Kidney International|July 5, 2017
Circulating complement factor H-related proteins 1 and 5 correlate with disease activity in IgA nephropathyNicholas R Medjeral-Thomas, Hannah J Lomax-Browne, Hannah Beckwith, et al.
Kidney International|May 17, 2017
Patients double-seropositive for ANCA and anti-GBM antibodies have varied renal survival, frequency of relapse, and outcomes compared to single-seropositive patientsStephen P McAdoo, Anisha Tanna, Zdenka Hrušková, et al.
Pageof 38