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D R Borchelt

Showing results (21-30 of 65) with videos related to

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Current Opinion in Neurobiology|October 1, 1996
Transgenic models of neurodegenerative diseasesM K Lee, D R Borchelt, P C Wong, et al.
The Journal of Biological Chemistry|September 26, 1997
Endoproteolytic processing and stabilization of wild-type and mutant presenilinT Ratovitski, H H Slunt, G Thinakaran, et al.
Biomolecular Engineering|May 5, 2001
Co-expression of multiple transgenes in mouse CNS: a comparison of strategiesJ L Jankowsky, H H Slunt, T Ratovitski, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 1, 1990
Acquisition of protease resistance by prion proteins in scrapie-infected cells does not require asparagine-linked glycosylationA Taraboulos, M Rogers, D R Borchelt, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 14, 1995
Superoxide dismutase is an abundant component in cell bodies, dendrites, and axons of motor neurons and in a subset of other neuronsC A Pardo, Z Xu, D R Borchelt, et al.
The Journal of Biological Chemistry|May 20, 1994
Rapid anterograde axonal transport of the cellular prion glycoprotein in the peripheral and central nervous systemsD R Borchelt, V E Koliatsos, M Guarnieri, et al.
Neurobiology of Disease|February 15, 2000
Enhanced synaptic potentiation in transgenic mice expressing presenilin 1 familial Alzheimer's disease mutation is normalized with a benzodiazepineS H Zaman, A Parent, A Laskey, et al.
Brain Pathology (Zurich, Switzerland)|October 1, 1996
Inherited neurodegenerative diseases and transgenic modelsD L Price, M W Becher, P C Wong, et al.
Human Molecular Genetics|July 13, 1999
Variation in the biochemical/biophysical properties of mutant superoxide dismutase 1 enzymes and the rate of disease progression in familial amyotrophic lateral sclerosis kindredsT Ratovitski, L B Corson, J Strain, et al.
Neurobiology of Disease|July 17, 1998
Stable association of presenilin derivatives and absence of presenilin interactions with APPG Thinakaran, J B Regard, C M Bouton, et al.
Pageof 7

Showing results (21-30 of 65) with videos related to

Sort By:
Pageof 7
Current Opinion in Neurobiology|October 1, 1996
Transgenic models of neurodegenerative diseasesM K Lee, D R Borchelt, P C Wong, et al.
The Journal of Biological Chemistry|September 26, 1997
Endoproteolytic processing and stabilization of wild-type and mutant presenilinT Ratovitski, H H Slunt, G Thinakaran, et al.
Biomolecular Engineering|May 5, 2001
Co-expression of multiple transgenes in mouse CNS: a comparison of strategiesJ L Jankowsky, H H Slunt, T Ratovitski, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 1, 1990
Acquisition of protease resistance by prion proteins in scrapie-infected cells does not require asparagine-linked glycosylationA Taraboulos, M Rogers, D R Borchelt, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 14, 1995
Superoxide dismutase is an abundant component in cell bodies, dendrites, and axons of motor neurons and in a subset of other neuronsC A Pardo, Z Xu, D R Borchelt, et al.
The Journal of Biological Chemistry|May 20, 1994
Rapid anterograde axonal transport of the cellular prion glycoprotein in the peripheral and central nervous systemsD R Borchelt, V E Koliatsos, M Guarnieri, et al.
Neurobiology of Disease|February 15, 2000
Enhanced synaptic potentiation in transgenic mice expressing presenilin 1 familial Alzheimer's disease mutation is normalized with a benzodiazepineS H Zaman, A Parent, A Laskey, et al.
Brain Pathology (Zurich, Switzerland)|October 1, 1996
Inherited neurodegenerative diseases and transgenic modelsD L Price, M W Becher, P C Wong, et al.
Human Molecular Genetics|July 13, 1999
Variation in the biochemical/biophysical properties of mutant superoxide dismutase 1 enzymes and the rate of disease progression in familial amyotrophic lateral sclerosis kindredsT Ratovitski, L B Corson, J Strain, et al.
Neurobiology of Disease|July 17, 1998
Stable association of presenilin derivatives and absence of presenilin interactions with APPG Thinakaran, J B Regard, C M Bouton, et al.
Pageof 7