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D R Borchelt

Showing results (41-50 of 65) with videos related to

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The Journal of Biological Chemistry|February 17, 1995
Superoxide dismutase 1 subunits with mutations linked to familial amyotrophic lateral sclerosis do not affect wild-type subunit functionD R Borchelt, M Guarnieri, P C Wong, et al.
Revue Neurologique|July 31, 1998
Amyotrophic lateral sclerosis and Alzheimer disease. Lessons from model systemsD L Price, P C Wong, D R Borchelt, et al.
Human Molecular Genetics|November 16, 2001
Hyper-expression of human apolipoprotein E4 in astroglia and neurons does not enhance amyloid deposition in transgenic miceC Lesuisse, G Xu, J Anderson, et al.
Neuron|June 1, 1995
An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondriaP C Wong, C A Pardo, D R Borchelt, et al.
DNA and Cell Biology|January 15, 2002
beta-Amyloid peptide vaccination results in marked changes in serum and brain Abeta levels in APPswe/PS1DeltaE9 mice, as detected by SELDI-TOF-based ProteinChip technologyA K Vehmas, D R Borchelt, D L Price, et al.
Human Molecular Genetics|September 25, 1997
Genetic modification of the phenotypes produced by amyloid precursor protein overexpression in transgenic miceG A Carlson, D R Borchelt, A Dake, et al.
Neuron|November 1, 1996
Protein topology of presenilin 1A Doan, G Thinakaran, D R Borchelt, et al.
Genetic Analysis : Biomolecular Engineering|December 1, 1996
A vector for expressing foreign genes in the brains and hearts of transgenic miceD R Borchelt, J Davis, M Fischer, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 16, 1994
Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activityD R Borchelt, M K Lee, H S Slunt, et al.
Neuron|January 6, 2001
Amyloid precursor proteins inhibit heme oxygenase activity and augment neurotoxicity in Alzheimer's diseaseM Takahashi, S Doré, C D Ferris, et al.
Pageof 7

Showing results (41-50 of 65) with videos related to

Sort By:
Pageof 7
The Journal of Biological Chemistry|February 17, 1995
Superoxide dismutase 1 subunits with mutations linked to familial amyotrophic lateral sclerosis do not affect wild-type subunit functionD R Borchelt, M Guarnieri, P C Wong, et al.
Revue Neurologique|July 31, 1998
Amyotrophic lateral sclerosis and Alzheimer disease. Lessons from model systemsD L Price, P C Wong, D R Borchelt, et al.
Human Molecular Genetics|November 16, 2001
Hyper-expression of human apolipoprotein E4 in astroglia and neurons does not enhance amyloid deposition in transgenic miceC Lesuisse, G Xu, J Anderson, et al.
Neuron|June 1, 1995
An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondriaP C Wong, C A Pardo, D R Borchelt, et al.
DNA and Cell Biology|January 15, 2002
beta-Amyloid peptide vaccination results in marked changes in serum and brain Abeta levels in APPswe/PS1DeltaE9 mice, as detected by SELDI-TOF-based ProteinChip technologyA K Vehmas, D R Borchelt, D L Price, et al.
Human Molecular Genetics|September 25, 1997
Genetic modification of the phenotypes produced by amyloid precursor protein overexpression in transgenic miceG A Carlson, D R Borchelt, A Dake, et al.
Neuron|November 1, 1996
Protein topology of presenilin 1A Doan, G Thinakaran, D R Borchelt, et al.
Genetic Analysis : Biomolecular Engineering|December 1, 1996
A vector for expressing foreign genes in the brains and hearts of transgenic miceD R Borchelt, J Davis, M Fischer, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 16, 1994
Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activityD R Borchelt, M K Lee, H S Slunt, et al.
Neuron|January 6, 2001
Amyloid precursor proteins inhibit heme oxygenase activity and augment neurotoxicity in Alzheimer's diseaseM Takahashi, S Doré, C D Ferris, et al.
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