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Eneuro|May 11, 2021
Templated α-Synuclein Inclusion Formation Is Independent of Endogenous TauLindsay E Stoyka, Casey L Mahoney, Drake R Thrasher, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 22, 2013
Dissociation of frontotemporal dementia-related deficits and neuroinflammation in progranulin haploinsufficient miceAnthony J Filiano, Lauren Herl Martens, Allen H Young, et al.
Science Translational Medicine|January 17, 2020
β-amyloid redirects norepinephrine signaling to activate the pathogenic GSK3β/tau cascadeFang Zhang, Mary Gannon, Yunjia Chen, et al.
Plos One|November 13, 2012
Usp14 deficiency increases tau phosphorylation without altering tau degradation or causing tau-dependent deficitsYoungnam N Jin, Ping-Chung Chen, Jennifer A Watson, et al.
Frontiers in Aging Neuroscience|September 19, 2012
Challenges and opportunities for characterizing cognitive aging across speciesErik D Roberson, R Anthony Defazio, Carol A Barnes, et al.
Annals of Clinical and Translational Neurology|November 16, 2020
Elevated levels of extracellular vesicles in progranulin-deficient mice and FTD-GRN PatientsAndrew E Arrant, Skylar E Davis, Rachael M Vollmer, et al.
Neurobiology of Disease|July 9, 2025
Reduction of sphingomyelinase activity associated with progranulin deficiency and frontotemporal dementiaNicholas R Boyle, Stephanie N Fox, Aniketh S Tadepalli, et al.
Acta Neuropathologica Communications|December 25, 2019
Impaired β-glucocerebrosidase activity and processing in frontotemporal dementia due to progranulin mutationsAndrew E Arrant, Jonathan R Roth, Nicholas R Boyle, et al.
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