Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

D Rudnicki

Showing results (1-10 of 29) with videos related to

Pageof 3
Sort By:
Expert Reviews in Molecular Medicine|October 31, 2003
Repeat expansion and autosomal dominant neurodegenerative disorders: consensus and controversyDobrila D Rudnicki, Russell L Margolis
Current Opinion in Neurology|November 3, 2016
Pathogenic insights from Huntington's disease-like 2 and other Huntington's disease genocopiesRussell L Margolis, Dobrila D Rudnicki
The Journal of Rheumatology|September 1, 1975
The efficacy of antimalarials in systemic lupus erythematosusR D Rudnicki, G E Gresham, N F Rothfield
Acta Neurologica Taiwanica|April 20, 2005
Huntington's disease like-2: review and updateRussell L Margolis, Dobrila D Rudnicki, Susan E Holmes
Human Molecular Genetics|June 16, 2011
A natural antisense transcript at the Huntington's disease repeat locus regulates HTT expressionDaniel W Chung, Dobrila D Rudnicki, Lan Yu, et al.
Frontiers in Robotics and AI|January 27, 2021
Exploring Novel Biologically-Relevant Chemical Space Through Artificial Intelligence: The NCATS ASPIRE ProgramKatharine K Duncan, Dobrila D Rudnicki, Christopher P Austin, et al.
Human Molecular Genetics|October 1, 1996
Idiopathic torsion dystonia: assignment of a gene to chromosome 18p in a German family with adult onset, autosomal dominant inheritance and purely focal distributionB Leube, D Rudnicki, T Ratzlaff, et al.
Journal of Neuropathology and Experimental Neurology|April 2, 2008
A comparison of huntington disease and huntington disease-like 2 neuropathologyDobrila D Rudnicki, Olga Pletnikova, Jean-Paul G Vonsattel, et al.
Biochemical and Biophysical Research Communications|January 27, 1999
Identification of the physiological promoter for spinocerebellar ataxia 2 gene reveals a CpG island for promoter activity situated into the exon 1 of this gene and provides data about the origin of the nonmethylated state of these types of islandsJ Aguiar, S Santurlidis, J Nowok, et al.
Annals of Neurology|March 28, 2007
Huntington's disease--like 2 is associated with CUG repeat-containing RNA fociDobrila D Rudnicki, Susan E Holmes, Mark W Lin, et al.
Pageof 3

Showing results (1-10 of 29) with videos related to

Sort By:
Pageof 3
Expert Reviews in Molecular Medicine|October 31, 2003
Repeat expansion and autosomal dominant neurodegenerative disorders: consensus and controversyDobrila D Rudnicki, Russell L Margolis
Current Opinion in Neurology|November 3, 2016
Pathogenic insights from Huntington's disease-like 2 and other Huntington's disease genocopiesRussell L Margolis, Dobrila D Rudnicki
The Journal of Rheumatology|September 1, 1975
The efficacy of antimalarials in systemic lupus erythematosusR D Rudnicki, G E Gresham, N F Rothfield
Acta Neurologica Taiwanica|April 20, 2005
Huntington's disease like-2: review and updateRussell L Margolis, Dobrila D Rudnicki, Susan E Holmes
Human Molecular Genetics|June 16, 2011
A natural antisense transcript at the Huntington's disease repeat locus regulates HTT expressionDaniel W Chung, Dobrila D Rudnicki, Lan Yu, et al.
Frontiers in Robotics and AI|January 27, 2021
Exploring Novel Biologically-Relevant Chemical Space Through Artificial Intelligence: The NCATS ASPIRE ProgramKatharine K Duncan, Dobrila D Rudnicki, Christopher P Austin, et al.
Human Molecular Genetics|October 1, 1996
Idiopathic torsion dystonia: assignment of a gene to chromosome 18p in a German family with adult onset, autosomal dominant inheritance and purely focal distributionB Leube, D Rudnicki, T Ratzlaff, et al.
Journal of Neuropathology and Experimental Neurology|April 2, 2008
A comparison of huntington disease and huntington disease-like 2 neuropathologyDobrila D Rudnicki, Olga Pletnikova, Jean-Paul G Vonsattel, et al.
Biochemical and Biophysical Research Communications|January 27, 1999
Identification of the physiological promoter for spinocerebellar ataxia 2 gene reveals a CpG island for promoter activity situated into the exon 1 of this gene and provides data about the origin of the nonmethylated state of these types of islandsJ Aguiar, S Santurlidis, J Nowok, et al.
Annals of Neurology|March 28, 2007
Huntington's disease--like 2 is associated with CUG repeat-containing RNA fociDobrila D Rudnicki, Susan E Holmes, Mark W Lin, et al.
Pageof 3