D Schmechel - JoVE Visualize

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Epilepsia|January 1, 1994

Complex genetic disease: can genetic strategies in Alzheimer's disease and new genetic mechanisms be applied to epilepsy?A D Roses, M A Pericak-Vance, A M Saunders, et al.

Journal of Immunological Methods|December 9, 2003

Limitations of monoclonal antibodies for monitoring of fungal aerosols using Penicillium brevicompactum as a model fungusD Schmechel, R L Górny, J P Simpson, et al.

Proceedings of the National Academy of Sciences of the United States of America|March 1, 1993

Apolipoprotein E: high-avidity binding to beta-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer diseaseW J Strittmatter, A M Saunders, D Schmechel, et al.

Neuroreport|April 29, 1998

Endogenous apolipoprotein E suppresses LPS-stimulated microglial nitric oxide productionD T Laskowitz, W D Matthew, E R Bennett, et al.

Journal of Neural Transmission. Supplementum|January 1, 1987

Failure of long term high-dose lecithin to retard progression of early-onset Alzheimer's diseaseA Heyman, D Schmechel, W Wilkinson, et al.

Environmental Health Perspectives|February 15, 2001

Estuary-associated syndrome in North Carolina: an occupational prevalence studyM Swinker, D Koltai, J Wilkins, et al.

Annals of the New York Academy of Sciences|September 24, 1993

Sequestration of amyloid beta-peptideD Goldgaber, A I Schwarzman, R Bhasin, et al.

Annals of Neurology|November 1, 1983

Alzheimer's disease: genetic aspects and associated clinical disordersA Heyman, W E Wilkinson, B J Hurwitz, et al.

Neurobiology of Disease|March 17, 1999

Sialylated human apolipoprotein E (apoEs) is preferentially associated with neuron-enriched cultures from APOE transgenic miceP T Xu, D Schmechel, H L Qiu, et al.

The Journal of Clinical Investigation|August 1, 1994

Apolipoprotein E associates with beta amyloid peptide of Alzheimer's disease to form novel monofibrils. Isoform apoE4 associates more efficiently than apoE3D A Sanan, K H Weisgraber, S J Russell, et al.

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Showing results (11-20 of 24) with videos related to

Sort By:

Pageof 3

Epilepsia|January 1, 1994

Complex genetic disease: can genetic strategies in Alzheimer's disease and new genetic mechanisms be applied to epilepsy?A D Roses, M A Pericak-Vance, A M Saunders, et al.

Journal of Immunological Methods|December 9, 2003

Limitations of monoclonal antibodies for monitoring of fungal aerosols using Penicillium brevicompactum as a model fungusD Schmechel, R L Górny, J P Simpson, et al.

Proceedings of the National Academy of Sciences of the United States of America|March 1, 1993

Apolipoprotein E: high-avidity binding to beta-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer diseaseW J Strittmatter, A M Saunders, D Schmechel, et al.

Neuroreport|April 29, 1998

Endogenous apolipoprotein E suppresses LPS-stimulated microglial nitric oxide productionD T Laskowitz, W D Matthew, E R Bennett, et al.

Journal of Neural Transmission. Supplementum|January 1, 1987

Failure of long term high-dose lecithin to retard progression of early-onset Alzheimer's diseaseA Heyman, D Schmechel, W Wilkinson, et al.

Environmental Health Perspectives|February 15, 2001

Estuary-associated syndrome in North Carolina: an occupational prevalence studyM Swinker, D Koltai, J Wilkins, et al.

Annals of the New York Academy of Sciences|September 24, 1993

Sequestration of amyloid beta-peptideD Goldgaber, A I Schwarzman, R Bhasin, et al.

Annals of Neurology|November 1, 1983

Alzheimer's disease: genetic aspects and associated clinical disordersA Heyman, W E Wilkinson, B J Hurwitz, et al.

Neurobiology of Disease|March 17, 1999

Sialylated human apolipoprotein E (apoEs) is preferentially associated with neuron-enriched cultures from APOE transgenic miceP T Xu, D Schmechel, H L Qiu, et al.

The Journal of Clinical Investigation|August 1, 1994

Apolipoprotein E associates with beta amyloid peptide of Alzheimer's disease to form novel monofibrils. Isoform apoE4 associates more efficiently than apoE3D A Sanan, K H Weisgraber, S J Russell, et al.

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