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The American Journal of Physiology|March 5, 1998
Hyperexpression of recombinant CFTR in heterologous cells alters its physiological propertiesR Mohammad-Panah, S Demolombe, D Riochet, et al.
The Journal of Pharmacology and Experimental Therapeutics|July 20, 1999
Interspecies differences in the cardiac negative inotropic effects of beta(3)-adrenoceptor agonistsC Gauthier, G Tavernier, J N Trochu, et al.
European Heart Journal|February 24, 2001
Dynamic analysis of the QT interval in long QT1 syndrome patients with a normal phenotypeG Lande, F Kyndt, I Baró, et al.
Biochemical and Biophysical Research Communications|August 14, 2001
Inactivating properties of recombinant ROMK2 channels expressed in mammalian cellsD F Riochet, R Mohammad-Panah, S C Hebert, et al.
Gene Therapy|August 1, 1996
A method for the rapid detection of recombinant CFTR during gene therapy in cystic fibrosisS Demolombe, I Baró, Z Bebok, et al.
American Journal of Human Genetics|March 26, 1999
Mutations in a dominant-negative isoform correlate with phenotype in inherited cardiac arrhythmiasR Mohammad-Panah, S Demolombe, N Neyroud, et al.
American Journal of Physiology. Cell Physiology|February 24, 2001
Differential expression of KvLQT1 and its regulator IsK in mouse epitheliaS Demolombe, D Franco, P de Boer, et al.
Cardiovascular Research|September 15, 2001
Divergent expression of delayed rectifier K(+) channel subunits during mouse heart developmentD Franco, S Demolombe, S Kupershmidt, et al.
The Journal of Biological Chemistry|April 18, 1998
A dominant negative isoform of the long QT syndrome 1 gene productS Demolombe, I Baró, Y Péréon, et al.
Circulation|December 19, 2001
Novel SCN5A mutation leading either to isolated cardiac conduction defect or Brugada syndrome in a large French familyF Kyndt, V Probst, F Potet, et al.
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