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D V Serreze

Showing results (1-10 of 60) with videos related to

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FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 1, 1993
Autoimmune diabetes results from genetic defects manifest by antigen presenting cellsD V Serreze
Current Directions in Autoimmunity|September 25, 2001
Genes and cellular requirements for autoimmune diabetes susceptibility in nonobese diabetic miceD V Serreze, E H Leiter
Regional Immunology|September 1, 1992
Antigen presenting cells and the immunogenetics of autoimmune diabetes in NOD miceE H Leiter, D V Serreze
Journal of Immunology (Baltimore, Md. : 1950)|June 1, 1988
Defective activation of T suppressor cell function in nonobese diabetic mice. Potential relation to cytokine deficienciesD V Serreze, E H Leiter
Current Opinion in Immunology|December 1, 1994
Genetic and pathogenic basis of autoimmune diabetes in NOD miceD V Serreze, E H Leiter
Journal of Immunology (Baltimore, Md. : 1950)|August 15, 1991
Development of diabetogenic T cells from NOD/Lt marrow is blocked when an allo-H-2 haplotype is expressed on cells of hemopoietic origin, but not on thymic epitheliumD V Serreze, E H Leiter
Clinical Immunology and Immunopathology|June 1, 1991
Autoimmune diabetes in the nonobese diabetic mouse: suppression of immune defects by bone marrow transplantation and implications for therapyE H Leiter, D V Serreze
Journal of Immunology (Baltimore, Md. : 1950)|July 22, 1999
Induction of glutamic acid decarboxylase 65-specific Th2 cells and suppression of autoimmune diabetes at late stages of disease is epitope dependentR Tisch, B Wang, D V Serreze
Journal of Autoimmunity|December 1, 1989
Immunostimulation circumvents diabetes in NOD/Lt miceD V Serreze, K Hamaguchi, E H Leiter
Immunology Today|May 1, 1990
The genetics and epidemiology of diabetes in NOD miceE H Leiter, D V Serreze, M Prochazka
Pageof 6

Showing results (1-10 of 60) with videos related to

Sort By:
Pageof 6
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 1, 1993
Autoimmune diabetes results from genetic defects manifest by antigen presenting cellsD V Serreze
Current Directions in Autoimmunity|September 25, 2001
Genes and cellular requirements for autoimmune diabetes susceptibility in nonobese diabetic miceD V Serreze, E H Leiter
Regional Immunology|September 1, 1992
Antigen presenting cells and the immunogenetics of autoimmune diabetes in NOD miceE H Leiter, D V Serreze
Journal of Immunology (Baltimore, Md. : 1950)|June 1, 1988
Defective activation of T suppressor cell function in nonobese diabetic mice. Potential relation to cytokine deficienciesD V Serreze, E H Leiter
Current Opinion in Immunology|December 1, 1994
Genetic and pathogenic basis of autoimmune diabetes in NOD miceD V Serreze, E H Leiter
Journal of Immunology (Baltimore, Md. : 1950)|August 15, 1991
Development of diabetogenic T cells from NOD/Lt marrow is blocked when an allo-H-2 haplotype is expressed on cells of hemopoietic origin, but not on thymic epitheliumD V Serreze, E H Leiter
Clinical Immunology and Immunopathology|June 1, 1991
Autoimmune diabetes in the nonobese diabetic mouse: suppression of immune defects by bone marrow transplantation and implications for therapyE H Leiter, D V Serreze
Journal of Immunology (Baltimore, Md. : 1950)|July 22, 1999
Induction of glutamic acid decarboxylase 65-specific Th2 cells and suppression of autoimmune diabetes at late stages of disease is epitope dependentR Tisch, B Wang, D V Serreze
Journal of Autoimmunity|December 1, 1989
Immunostimulation circumvents diabetes in NOD/Lt miceD V Serreze, K Hamaguchi, E H Leiter
Immunology Today|May 1, 1990
The genetics and epidemiology of diabetes in NOD miceE H Leiter, D V Serreze, M Prochazka
Pageof 6