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Plos One|October 19, 2017
Functional improvement of dystrophic muscle by repression of utrophin: let-7c interactionManoj K Mishra, Emanuele Loro, Kasturi Sengupta, et al.
Gene Therapy|May 26, 2006
Antisense oligonucleotide-induced exon skipping restores dystrophin expression in vitro in a canine model of DMDG McClorey, H M Moulton, P L Iversen, et al.
International Journal of Molecular Sciences|July 8, 2020
Single Exon Skipping Can Address a Multi-Exon Duplication in the Dystrophin GeneKane Greer, Russell Johnsen, Yoram Nevo, et al.
Molecular Therapy. Nucleic Acids|December 17, 2017
Rational Design of Short Locked Nucleic Acid-Modified 2'-O-Methyl Antisense Oligonucleotides for Efficient Exon-Skipping In VitroBao T Le, Abbie M Adams, Susan Fletcher, et al.
Neuromuscular Disorders : NMD|September 7, 2010
Personalized exon skipping strategies to address clustered non-deletion dystrophin mutationsSarah Forrest, Penny L Meloni, Francesco Muntoni, et al.
FEBS Letters|October 7, 2003
Enhanced in vivo delivery of antisense oligonucleotides to restore dystrophin expression in adult mdx mouse muscleK E Wells, S Fletcher, C J Mann, et al.
Genetic Vaccines and Therapy|May 25, 2006
Induction of revertant fibres in the mdx mouse using antisense oligonucleotidesAbbie M Fall, Russell Johnsen, Kaite Honeyman, et al.
Experimental Neurology|June 26, 2001
The regrowth of axons within tissue defects in the CNS is promoted by implanted hydrogel matrices that contain BDNF and CNTF producing fibroblastsN K Loh, S Woerly, S M Bunt, et al.
Neuromuscular Disorders : NMD|August 9, 2005
Terminal antisense oligonucleotide modifications can enhance induced exon skippingBijanka L Gebski, Stephen J Errington, Russell D Johnsen, et al.
Plos One|January 5, 2012
Translational regulation of utrophin by miRNAsUtpal Basu, Olga Lozynska, Catherine Moorwood, et al.
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