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European Journal of Biochemistry|September 24, 1990
De-ADP-ribosylation actin by Clostridium perfringens iota-toxin and Clostridium botulinum C2 toxinI Just, U Geipel, A Wegner, et al.
Advances in Experimental Medicine and Biology|January 1, 1997
ADP-ribosylating and glucosylating toxins as tools to study secretion in RBL cellsU Prepens, I Just, F Hofmann, et al.
The Journal of Biological Chemistry|April 5, 2000
Recognition of RhoA by Clostridium botulinum C3 exoenzymeC Wilde, H Genth, K Aktories, et al.
Biochemical and Biophysical Research Communications|October 3, 1996
The ras-related protein Ral is monoglucosylated by Clostridium sordellii lethal toxinF Hofmann, G Rex, K Aktories, et al.
The Journal of Biological Chemistry|June 20, 1998
Functional consequences of monoglucosylation of Ha-Ras at effector domain amino acid threonine 35C Herrmann, M R Ahmadian, F Hofmann, et al.
The Journal of Cell Biology|September 1, 1989
Influence of botulinum C2 toxin on F-actin and N-formyl peptide receptor dynamics in human neutrophilsJ Norgauer, I Just, K Aktories, et al.
Journal of Chromatography. B, Analytical Technologies in the Biomedical and Life Sciences|December 26, 2008
Identification of biomarkers indicating cellular changes after treatment of neuronal cells with the C3 exoenzyme from Clostridium botulinum using the iTRAQ protocol and LC-MS/MS analysisM V Muetzelburg, F Hofmann, I Just, et al.
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