Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Dale Abel

Showing results (91-100 of 266) with videos related to

Pageof 27
Sort By:
Hypertension (Dallas, Tex. : 1979)|November 4, 2009
Mammalian target of rapamycin is a critical regulator of cardiac hypertrophy in spontaneously hypertensive ratsWill Soesanto, Han-Yi Lin, Eric Hu, et al.
Journal of Molecular and Cellular Cardiology|October 18, 2015
Phosphoinositide dependent protein kinase 1 is required for exercise-induced cardiac hypertrophy but not the associated mitochondrial adaptationsJunghyun Noh, Adam R Wende, Curtis D Olsen, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|April 30, 2014
Maintaining PGC-1α expression following pressure overload-induced cardiac hypertrophy preserves angiogenesis but not contractile or mitochondrial functionRenata O Pereira, Adam R Wende, Ashley Crum, et al.
Molecular Medicine (Cambridge, Mass.)|March 5, 2008
Iron-mediated inhibition of mitochondrial manganese uptake mediates mitochondrial dysfunction in a mouse model of hemochromatosisHani A Jouihan, Paul A Cobine, Robert C Cooksey, et al.
Journal of Cardiac Failure|June 10, 2006
Dilated cardiomyopathy resulting from high-level myocardial expression of Cre-recombinaseAntje Buerger, Olga Rozhitskaya, Megan C Sherwood, et al.
Plos One|August 8, 2012
HSPB2 is dispensable for the cardiac hypertrophic response but reduces mitochondrial energetics following pressure overload in miceTakahiro Ishiwata, András Orosz, Xiaohui Wang, et al.
American Journal of Physiology. Endocrinology and Metabolism|August 27, 2010
An APPL1-AMPK signaling axis mediates beneficial metabolic effects of adiponectin in the heartXiangping Fang, Rengasamy Palanivel, Justin Cresser, et al.
The Journal of Clinical Investigation|April 16, 2004
Targeted deletion of BMK1/ERK5 in adult mice perturbs vascular integrity and leads to endothelial failureMasaaki Hayashi, Sung-Woo Kim, Kyoko Imanaka-Yoshida, et al.
Cell Reports|July 27, 2017
Deletion of GLUT1 and GLUT3 Reveals Multiple Roles for Glucose Metabolism in Platelet and Megakaryocyte FunctionTrevor P Fidler, Robert A Campbell, Trevor Funari, et al.
Endocrinology|September 6, 2005
Reduced cardiac efficiency and altered substrate metabolism precedes the onset of hyperglycemia and contractile dysfunction in two mouse models of insulin resistance and obesityJonathan Buchanan, Pradip K Mazumder, Ping Hu, et al.
Pageof 27

Showing results (91-100 of 266) with videos related to

Sort By:
Pageof 27
Hypertension (Dallas, Tex. : 1979)|November 4, 2009
Mammalian target of rapamycin is a critical regulator of cardiac hypertrophy in spontaneously hypertensive ratsWill Soesanto, Han-Yi Lin, Eric Hu, et al.
Journal of Molecular and Cellular Cardiology|October 18, 2015
Phosphoinositide dependent protein kinase 1 is required for exercise-induced cardiac hypertrophy but not the associated mitochondrial adaptationsJunghyun Noh, Adam R Wende, Curtis D Olsen, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|April 30, 2014
Maintaining PGC-1α expression following pressure overload-induced cardiac hypertrophy preserves angiogenesis but not contractile or mitochondrial functionRenata O Pereira, Adam R Wende, Ashley Crum, et al.
Molecular Medicine (Cambridge, Mass.)|March 5, 2008
Iron-mediated inhibition of mitochondrial manganese uptake mediates mitochondrial dysfunction in a mouse model of hemochromatosisHani A Jouihan, Paul A Cobine, Robert C Cooksey, et al.
Journal of Cardiac Failure|June 10, 2006
Dilated cardiomyopathy resulting from high-level myocardial expression of Cre-recombinaseAntje Buerger, Olga Rozhitskaya, Megan C Sherwood, et al.
Plos One|August 8, 2012
HSPB2 is dispensable for the cardiac hypertrophic response but reduces mitochondrial energetics following pressure overload in miceTakahiro Ishiwata, András Orosz, Xiaohui Wang, et al.
American Journal of Physiology. Endocrinology and Metabolism|August 27, 2010
An APPL1-AMPK signaling axis mediates beneficial metabolic effects of adiponectin in the heartXiangping Fang, Rengasamy Palanivel, Justin Cresser, et al.
The Journal of Clinical Investigation|April 16, 2004
Targeted deletion of BMK1/ERK5 in adult mice perturbs vascular integrity and leads to endothelial failureMasaaki Hayashi, Sung-Woo Kim, Kyoko Imanaka-Yoshida, et al.
Cell Reports|July 27, 2017
Deletion of GLUT1 and GLUT3 Reveals Multiple Roles for Glucose Metabolism in Platelet and Megakaryocyte FunctionTrevor P Fidler, Robert A Campbell, Trevor Funari, et al.
Endocrinology|September 6, 2005
Reduced cardiac efficiency and altered substrate metabolism precedes the onset of hyperglycemia and contractile dysfunction in two mouse models of insulin resistance and obesityJonathan Buchanan, Pradip K Mazumder, Ping Hu, et al.
Pageof 27