Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Dale Abel

Showing results (181-190 of 266) with videos related to

Pageof 27
Sort By:
The Journal of Clinical Investigation|March 6, 2002
Insulin signaling coordinately regulates cardiac size, metabolism, and contractile protein isoform expressionDarrell D Belke, Sandrine Betuing, Martin J Tuttle, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 21, 2017
Glucose transporter 4-deficient hearts develop maladaptive hypertrophy in response to physiological or pathological stressesAdam R Wende, Jaetaek Kim, William L Holland, et al.
Autophagy|February 1, 2022
Perinatal versus adult loss of ULK1 and ULK2 distinctly influences cardiac autophagy and functionMatthew P Harris, Quan J Zhang, Cole T Cochran, et al.
Cell Metabolism|March 22, 2022
The glucose transporter GLUT3 controls T helper 17 cell responses through glycolytic-epigenetic reprogrammingSophia M Hochrein, Hao Wu, Miriam Eckstein, et al.
Journal of Molecular and Cellular Cardiology|February 21, 2012
Genetic loss of insulin receptors worsens cardiac efficiency in diabetesHeiko Bugger, Christian Riehle, Bharat Jaishy, et al.
The Journal of Biological Chemistry|December 26, 2012
Talin1 has unique expression versus talin 2 in the heart and modifies the hypertrophic response to pressure overloadAna Maria Manso, Ruixia Li, Susan J Monkley, et al.
Biomolecules|December 23, 2022
Muscle-Specific Ablation of Glucose Transporter 1 (GLUT1) Does Not Impair Basal or Overload-Stimulated Skeletal Muscle Glucose UptakeShawna L McMillin, Parker L Evans, William M Taylor, et al.
American Journal of Physiology. Heart and Circulatory Physiology|February 12, 2021
FoxO1 is required for physiological cardiac hypertrophy induced by exercise but not by constitutively active PI3KKate L Weeks, Yow Keat Tham, Suzan G Yildiz, et al.
Cardiovascular Research|December 3, 2011
Energy-preserving effects of IGF-1 antagonize starvation-induced cardiac autophagyRodrigo Troncoso, Jose Miguel Vicencio, Valentina Parra, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 29, 2013
IGF-1 receptor deficiency in thyrocytes impairs thyroid hormone secretion and completely inhibits TSH-stimulated goiterSangmi Ock, Jihyun Ahn, Seok Hong Lee, et al.
Pageof 27

Showing results (181-190 of 266) with videos related to

Sort By:
Pageof 27
The Journal of Clinical Investigation|March 6, 2002
Insulin signaling coordinately regulates cardiac size, metabolism, and contractile protein isoform expressionDarrell D Belke, Sandrine Betuing, Martin J Tuttle, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 21, 2017
Glucose transporter 4-deficient hearts develop maladaptive hypertrophy in response to physiological or pathological stressesAdam R Wende, Jaetaek Kim, William L Holland, et al.
Autophagy|February 1, 2022
Perinatal versus adult loss of ULK1 and ULK2 distinctly influences cardiac autophagy and functionMatthew P Harris, Quan J Zhang, Cole T Cochran, et al.
Cell Metabolism|March 22, 2022
The glucose transporter GLUT3 controls T helper 17 cell responses through glycolytic-epigenetic reprogrammingSophia M Hochrein, Hao Wu, Miriam Eckstein, et al.
Journal of Molecular and Cellular Cardiology|February 21, 2012
Genetic loss of insulin receptors worsens cardiac efficiency in diabetesHeiko Bugger, Christian Riehle, Bharat Jaishy, et al.
The Journal of Biological Chemistry|December 26, 2012
Talin1 has unique expression versus talin 2 in the heart and modifies the hypertrophic response to pressure overloadAna Maria Manso, Ruixia Li, Susan J Monkley, et al.
Biomolecules|December 23, 2022
Muscle-Specific Ablation of Glucose Transporter 1 (GLUT1) Does Not Impair Basal or Overload-Stimulated Skeletal Muscle Glucose UptakeShawna L McMillin, Parker L Evans, William M Taylor, et al.
American Journal of Physiology. Heart and Circulatory Physiology|February 12, 2021
FoxO1 is required for physiological cardiac hypertrophy induced by exercise but not by constitutively active PI3KKate L Weeks, Yow Keat Tham, Suzan G Yildiz, et al.
Cardiovascular Research|December 3, 2011
Energy-preserving effects of IGF-1 antagonize starvation-induced cardiac autophagyRodrigo Troncoso, Jose Miguel Vicencio, Valentina Parra, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 29, 2013
IGF-1 receptor deficiency in thyrocytes impairs thyroid hormone secretion and completely inhibits TSH-stimulated goiterSangmi Ock, Jihyun Ahn, Seok Hong Lee, et al.
Pageof 27