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Hormone Research in Paediatrics|December 14, 2011
The genetics of 3-M syndrome: unravelling a potential new regulatory growth pathwayDan Hanson, Philip G Murray, Graeme C M Black, et al.Journal of Molecular Endocrinology|April 9, 2014
Identifying biological pathways that underlie primordial short stature using network analysisDan Hanson, Adam Stevens, Philip G Murray, et al.Veterinary Microbiology|November 27, 2019
Comparison of the sensitivity of laryngeal swabs and deep tracheal catheters for detection of Mycoplasma hyopneumoniae in experimentally and naturally infected pigs early and late after infectionAmanda Sponheim, Julio Alvarez, Eduardo Fano, et al.Plos One|December 7, 2018
A randomized controlled trial to evaluate performance of pigs raised in antibiotic-free or conventional production systems following challenge with porcine reproductive and respiratory syndrome virusScott Dee, Jose Ezequiel Guzman, Dan Hanson, et al.Transboundary and Emerging Diseases|July 25, 2020
An evaluation of additives for mitigating the risk of virus-contaminated feed using an ice-block challenge modelScott A Dee, Megan C Niederwerder, Roy Edler, et al.Transboundary and Emerging Diseases|March 25, 2021
Evidence of viral survival in representative volumes of feed and feed ingredients during long-distance commercial transport across the continental United StatesScott Dee, Apoorva Shah, Cassandra Jones, et al.Clinical Endocrinology|May 26, 2012
Exploring the spectrum of 3-M syndrome, a primordial short stature disorder of disrupted ubiquitinationPeter E Clayton, Dan Hanson, Lucia Magee, et al.American Journal of Human Genetics|June 2, 2009
The primordial growth disorder 3-M syndrome connects ubiquitination to the cytoskeletal adaptor OBSL1Dan Hanson, Philip G Murray, Amit Sud, et al.The Journal of Clinical Endocrinology and Metabolism|March 6, 2015
An XRCC4 splice mutation associated with severe short stature, gonadal failure, and early-onset metabolic syndromeChristiaan de Bruin, Verónica Mericq, Shayne F Andrew, et al.American Journal of Human Genetics|July 9, 2011
Exome sequencing identifies CCDC8 mutations in 3-M syndrome, suggesting that CCDC8 contributes in a pathway with CUL7 and OBSL1 to control human growthDan Hanson, Philip G Murray, James O'Sullivan, et al.Pageof 2