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The American Journal of Pathology|March 2, 2006
Tumor necrosis factor-alpha mediates diabetes-enhanced apoptosis of matrix-producing cells and impairs diabetic healingRongkun Liu, Harbinder S Bal, Tesfahun Desta, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 5, 2008
The transcription factor ST18 regulates proapoptotic and proinflammatory gene expression in fibroblastsJulia Yang, Michelle F Siqueira, Yugal Behl, et al.
Circulation|September 9, 2004
Interleukin-1 receptor signaling mediates atherosclerosis associated with bacterial exposure and/or a high-fat diet in a murine apolipoprotein E heterozygote model: pharmacotherapeutic implicationsHunghui Chi, Emmanuel Messas, Robert A Levine, et al.
Bone|March 25, 2019
Deletion of FOXO1 in chondrocytes rescues the effect of diabetes on mechanical strength in fracture healingYongjian Lu, Mohammed Alharbi, Citong Zhang, et al.
The American Journal of Pathology|April 12, 2008
Diabetes-enhanced tumor necrosis factor-alpha production promotes apoptosis and the loss of retinal microvascular cells in type 1 and type 2 models of diabetic retinopathyYugal Behl, Padmaja Krothapalli, Tesfahun Desta, et al.
Critical Reviews in Immunology|February 13, 2018
The Role of Forkhead Box 1 (FOXO1) in the Immune System: Dendritic Cells, T Cells, B Cells, and Hematopoietic Stem CellsAdriana Alicia Cabrera-Ortega, Daniel Feinberg, Youde Liang, et al.
Endocrinology|July 31, 2004
Influence of diabetes on the exacerbation of an inflammatory response in cardiovascular tissueHuafei Lu, Markos Raptis, Elena Black, et al.
The Journal of Cell Biology|October 23, 2013
FOXO1 promotes wound healing through the up-regulation of TGF-β1 and prevention of oxidative stressBhaskar Ponugoti, Fanxing Xu, Chenying Zhang, et al.
The Journal of Biological Chemistry|October 11, 2003
Lipopolysaccharides indirectly stimulate apoptosis and global induction of apoptotic genes in fibroblastsMani Alikhani, Zoubin Alikhani, Hongbing He, et al.
Endocrinology|October 4, 2003
Diabetes causes decreased osteoclastogenesis, reduced bone formation, and enhanced apoptosis of osteoblastic cells in bacteria stimulated bone lossHongbing He, Rongkun Liu, Tesfahun Desta, et al.
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