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American Journal of Respiratory Cell and Molecular Biology|April 4, 2019
The Innate Immune Protein S100A9 Protects from T-Helper Cell Type 2-mediated Allergic Airway InflammationLauren D Palmer, K Nichole Maloney, Kelli L Boyd, et al.Journal of Virology|February 23, 2023
Influence of Sex on Respiratory Syncytial Virus Genotype Infection Frequency and Nasopharyngeal MicrobiomeYi Tan, Meghan H Shilts, Christian Rosas-Salazar, et al.The Journal of Allergy and Clinical Immunology|June 27, 2022
Human IgE mAbs identify major antigens of parasitic worm infectionAzadeh Hadadianpour, Jacob Daniel, Jian Zhang, et al.Journal of Immunology (Baltimore, Md. : 1950)|January 24, 2007
Synthetic prostacyclin analogs differentially regulate macrophage function via distinct analog-receptor binding specificitiesDavid M Aronoff, Camila M Peres, Carlos H Serezani, et al.American Journal of Respiratory Cell and Molecular Biology|February 9, 2013
IL-17A induces signal transducers and activators of transcription-6-independent airway mucous cell metaplasiaDawn C Newcomb, Madison G Boswell, Taylor P Sherrill, et al.The Journal of Infectious Diseases|March 3, 2026
Infant Infection With Respiratory Syncytial Virus Genotypes and Subsequent Childhood Asthma RiskChristian Rosas-Salazar, Tebeb Gebretsadik, James D Chappell, et al.Cancer|February 24, 2023
Applying a risk prediction model for bloodstream infection in a febrile, nonseverely neutropenic cohort of pediatric stem cell transplant patientsKasey Jackson, Victoria Anderson, Zhiguo Zhao, et al.Frontiers in Pediatrics|August 27, 2021
Case Report: Immune Dysregulation Due to Toxoplasma gondii Reactivation After Allogeneic Hematopoietic Cell TransplantRobert B Lindell, Michael S Wolf, Alicia M Alcamo, et al.Journal of Immunology (Baltimore, Md. : 1950)|June 4, 2017
STAT1 Represses Cytokine-Producing Group 2 and Group 3 Innate Lymphoid Cells during Viral InfectionMatthew T Stier, Kasia Goleniewska, Jacqueline Y Cephus, et al.Journal of Immunology (Baltimore, Md. : 1950)|December 13, 2005
Allergen-induced airway hyperresponsiveness mediated by cyclooxygenase inhibition is not dependent on 5-lipoxygenase or IL-5, but is IL-13 dependentR Stokes Peebles, Koichi Hashimoto, James R Sheller, et al.Pageof 21