Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Daniel Hohl

Showing results (61-70 of 101) with videos related to

Pageof 11
Sort By:
EMBO Molecular Medicine|March 3, 2012
Nrf2 links epidermal barrier function with antioxidant defenseMatthias Schäfer, Hany Farwanah, Ann-Helen Willrodt, et al.
EMBO Molecular Medicine|October 9, 2023
NRF3 suppresses squamous carcinogenesis, involving the unfolded protein response regulator HSPA5Selina Gurri, Beat Siegenthaler, Michael Cangkrama, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 15, 2013
Mast cells are dispensable for normal and activin-promoted wound healing and skin carcinogenesisMaria Antsiferova, Caroline Martin, Marcel Huber, et al.
The Journal of Investigative Dermatology|July 23, 2013
UVB-induced skin inflammation and cutaneous tissue injury is dependent on the MHC class I-like protein, CD1dStephan Ryser, Marlène Schuppli, Beatrice Gauthier, et al.
Human Molecular Genetics|March 1, 2006
A human keratin 10 knockout causes recessive epidermolytic hyperkeratosisFelix B Müller, Marcel Huber, Tamar Kinaciyan, et al.
Pediatric Dermatology|March 19, 2022
Evaluation of neurodevelopmental symptoms in 10 cases of neonatal ichthyosis and sclerosing cholangitis syndromeDeborah Salik, Smail Hadj-Rabia, Daniel Hohl, et al.
The Journal of Investigative Dermatology|September 30, 2006
SLURP1 is a late marker of epidermal differentiation and is absent in Mal de MeledaBertrand Favre, Laure Plantard, Lorène Aeschbach, et al.
The Journal of Investigative Dermatology|February 20, 2002
Deletion of the cytoplasmatic domain of BP180/collagen XVII causes a phenotype with predominant features of epidermolysis bullosa simplexMarcel Huber, Michaela Floeth, Luca Borradori, et al.
Journal of Drugs in Dermatology : JDD|April 14, 2021
Treatment of Impetigo and Antimicrobial ResistanceLawrence A Schachner, Charles W Lynde, Leon H Kircik, et al.
Nature|April 4, 2008
Cutaneous cancer stem cell maintenance is dependent on beta-catenin signallingIlaria Malanchi, Hector Peinado, Deepika Kassen, et al.
Pageof 11

Showing results (61-70 of 101) with videos related to

Sort By:
Pageof 11
EMBO Molecular Medicine|March 3, 2012
Nrf2 links epidermal barrier function with antioxidant defenseMatthias Schäfer, Hany Farwanah, Ann-Helen Willrodt, et al.
EMBO Molecular Medicine|October 9, 2023
NRF3 suppresses squamous carcinogenesis, involving the unfolded protein response regulator HSPA5Selina Gurri, Beat Siegenthaler, Michael Cangkrama, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 15, 2013
Mast cells are dispensable for normal and activin-promoted wound healing and skin carcinogenesisMaria Antsiferova, Caroline Martin, Marcel Huber, et al.
The Journal of Investigative Dermatology|July 23, 2013
UVB-induced skin inflammation and cutaneous tissue injury is dependent on the MHC class I-like protein, CD1dStephan Ryser, Marlène Schuppli, Beatrice Gauthier, et al.
Human Molecular Genetics|March 1, 2006
A human keratin 10 knockout causes recessive epidermolytic hyperkeratosisFelix B Müller, Marcel Huber, Tamar Kinaciyan, et al.
Pediatric Dermatology|March 19, 2022
Evaluation of neurodevelopmental symptoms in 10 cases of neonatal ichthyosis and sclerosing cholangitis syndromeDeborah Salik, Smail Hadj-Rabia, Daniel Hohl, et al.
The Journal of Investigative Dermatology|September 30, 2006
SLURP1 is a late marker of epidermal differentiation and is absent in Mal de MeledaBertrand Favre, Laure Plantard, Lorène Aeschbach, et al.
The Journal of Investigative Dermatology|February 20, 2002
Deletion of the cytoplasmatic domain of BP180/collagen XVII causes a phenotype with predominant features of epidermolysis bullosa simplexMarcel Huber, Michaela Floeth, Luca Borradori, et al.
Journal of Drugs in Dermatology : JDD|April 14, 2021
Treatment of Impetigo and Antimicrobial ResistanceLawrence A Schachner, Charles W Lynde, Leon H Kircik, et al.
Nature|April 4, 2008
Cutaneous cancer stem cell maintenance is dependent on beta-catenin signallingIlaria Malanchi, Hector Peinado, Deepika Kassen, et al.
Pageof 11