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Daniel K Stachel

Showing results (1-10 of 4) with videos related to

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Transfusion|May 12, 2006
Platelet function in variable platelet split products intended for neonatal transfusionErwin F Strasser, Daniel K Stachel, Peter Schwarzkopf, et al.
Journal of Hematotherapy & Stem Cell Research|June 14, 2003
Successful stem cell mobilization with stem cell factor and granulocyte colony-stimulating factor in patients with solid tumors failing conventional mobilization with chemotherapy and G-CSFDaniel K Stachel, Alfred Leipold, Thorsten Krapf, et al.
Journal of Pediatric Hematology/Oncology|December 14, 2005
Simultaneous control of third-degree graft-versus-host disease and prevention of recurrence of juvenile myelomonocytic leukemia (JMML) with 6-mercaptopurine following fulminant JMML relapse early after KIR-mismatched bone marrow transplantationDaniel K Stachel, Alfred Leipold, Michaela Kuhlen, et al.
Human Mutation|December 3, 2009
The most frequent DCLRE1C (ARTEMIS) mutations are based on homologous recombination eventsUlrich Pannicke, Manfred Hönig, Ilka Schulze, et al.
Pageof 1

Showing results (1-10 of 4) with videos related to

Sort By:
Pageof 1
Transfusion|May 12, 2006
Platelet function in variable platelet split products intended for neonatal transfusionErwin F Strasser, Daniel K Stachel, Peter Schwarzkopf, et al.
Journal of Hematotherapy & Stem Cell Research|June 14, 2003
Successful stem cell mobilization with stem cell factor and granulocyte colony-stimulating factor in patients with solid tumors failing conventional mobilization with chemotherapy and G-CSFDaniel K Stachel, Alfred Leipold, Thorsten Krapf, et al.
Journal of Pediatric Hematology/Oncology|December 14, 2005
Simultaneous control of third-degree graft-versus-host disease and prevention of recurrence of juvenile myelomonocytic leukemia (JMML) with 6-mercaptopurine following fulminant JMML relapse early after KIR-mismatched bone marrow transplantationDaniel K Stachel, Alfred Leipold, Michaela Kuhlen, et al.
Human Mutation|December 3, 2009
The most frequent DCLRE1C (ARTEMIS) mutations are based on homologous recombination eventsUlrich Pannicke, Manfred Hönig, Ilka Schulze, et al.
Pageof 1