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Daniel Mertens

Showing results (51-60 of 100) with videos related to

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Proceedings of the National Academy of Sciences of the United States of America|May 11, 2006
Allelic silencing at the tumor-suppressor locus 13q14.3 suggests an epigenetic tumor-suppressor mechanismDaniel Mertens, Stephan Wolf, Cordula Tschuch, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|May 4, 2005
Evidence for distinct pathomechanisms in genetic subgroups of chronic lymphocytic leukemia revealed by quantitative expression analysis of cell cycle, activation, and apoptosis-associated genesDirk L Kienle, Christian Korz, Beate Hosch, et al.
International Journal of Cancer|May 16, 2014
Loss of cooperativity of secreted CD40L and increased dose-response to IL4 on CLL cell viability correlates with enhanced activation of NF-kB and STAT6Nupur Bhattacharya, Michaela Reichenzeller, Maiwen Caudron-Herger, et al.
Clinical Epigenetics|October 27, 2023
Predictive value of DNA methylation patterns in AML patients treated with an azacytidine containing induction regimenMaximilian Schmutz, Manuela Zucknick, Richard F Schlenk, et al.
British Journal of Haematology|May 28, 2011
Nurse-like cells show deregulated expression of genes involved in immunocompetenceNupur Bhattacharya, Susanne Diener, Irina S Idler, et al.
International Journal of Cancer|May 9, 2022
Modeling the B-cell receptor signaling on single cell level reveals a stable network circuit topology between nonmalignant B cells and chronic lymphocytic leukemia cells and between untreated cells and cells treated with kinase inhibitorsChristine Wolf, Carsten Maus, Michael R O Persicke, et al.
Haematologica|April 16, 2016
Krüppel-like factor 4 (KLF4) inactivation in chronic lymphocytic leukemia correlates with promoter DNA-methylation and can be reversed by inhibition of NOTCH signalingKatharina Filarsky, Angela Garding, Natalia Becker, et al.
International Journal of Cancer|February 24, 2015
Epigenetic silencing of miR-708 enhances NF-κB signaling in chronic lymphocytic leukemiaConstance Baer, Christopher C Oakes, Amy S Ruppert, et al.
Haematologica|March 11, 2021
Clonal evolution in chronic lymphocytic leukemia is scant in relapsed but accelerated in refractory cases after chemo(immune) therapyMarc Zapatka, Eugen Tausch, Selcen Öztürk, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|January 7, 2006
Reduced expression of CAMTA1 correlates with adverse outcome in neuroblastoma patientsKai-Oliver Henrich, Matthias Fischer, Daniel Mertens, et al.
Pageof 10

Showing results (51-60 of 100) with videos related to

Sort By:
Pageof 10
Proceedings of the National Academy of Sciences of the United States of America|May 11, 2006
Allelic silencing at the tumor-suppressor locus 13q14.3 suggests an epigenetic tumor-suppressor mechanismDaniel Mertens, Stephan Wolf, Cordula Tschuch, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|May 4, 2005
Evidence for distinct pathomechanisms in genetic subgroups of chronic lymphocytic leukemia revealed by quantitative expression analysis of cell cycle, activation, and apoptosis-associated genesDirk L Kienle, Christian Korz, Beate Hosch, et al.
International Journal of Cancer|May 16, 2014
Loss of cooperativity of secreted CD40L and increased dose-response to IL4 on CLL cell viability correlates with enhanced activation of NF-kB and STAT6Nupur Bhattacharya, Michaela Reichenzeller, Maiwen Caudron-Herger, et al.
Clinical Epigenetics|October 27, 2023
Predictive value of DNA methylation patterns in AML patients treated with an azacytidine containing induction regimenMaximilian Schmutz, Manuela Zucknick, Richard F Schlenk, et al.
British Journal of Haematology|May 28, 2011
Nurse-like cells show deregulated expression of genes involved in immunocompetenceNupur Bhattacharya, Susanne Diener, Irina S Idler, et al.
International Journal of Cancer|May 9, 2022
Modeling the B-cell receptor signaling on single cell level reveals a stable network circuit topology between nonmalignant B cells and chronic lymphocytic leukemia cells and between untreated cells and cells treated with kinase inhibitorsChristine Wolf, Carsten Maus, Michael R O Persicke, et al.
Haematologica|April 16, 2016
Krüppel-like factor 4 (KLF4) inactivation in chronic lymphocytic leukemia correlates with promoter DNA-methylation and can be reversed by inhibition of NOTCH signalingKatharina Filarsky, Angela Garding, Natalia Becker, et al.
International Journal of Cancer|February 24, 2015
Epigenetic silencing of miR-708 enhances NF-κB signaling in chronic lymphocytic leukemiaConstance Baer, Christopher C Oakes, Amy S Ruppert, et al.
Haematologica|March 11, 2021
Clonal evolution in chronic lymphocytic leukemia is scant in relapsed but accelerated in refractory cases after chemo(immune) therapyMarc Zapatka, Eugen Tausch, Selcen Öztürk, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|January 7, 2006
Reduced expression of CAMTA1 correlates with adverse outcome in neuroblastoma patientsKai-Oliver Henrich, Matthias Fischer, Daniel Mertens, et al.
Pageof 10