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Science Advances|April 20, 2019
Secondary nucleation and elongation occur at different sites on Alzheimer's amyloid-β aggregatesTom Scheidt, Urszula Łapińska, Janet R Kumita, et al.ACS Nano|October 30, 2018
Quantifying Co-Oligomer Formation by α-SynucleinMarija Iljina, Alexander J Dear, Gonzalo A Garcia, et al.Acta Neuropathologica Communications|November 9, 2017
Clusterin protects neurons against intracellular proteotoxicityJenna M Gregory, Daniel R Whiten, Rebecca A Brown, et al.Acta Neuropathologica Communications|July 28, 2019
Soluble aggregates present in cerebrospinal fluid change in size and mechanism of toxicity during Alzheimer's disease progressionSuman De, Daniel R Whiten, Francesco S Ruggeri, et al.Chembiochem : a European Journal of Chemical Biology|July 28, 2018
Nanoscopic Characterisation of Individual Endogenous Protein Aggregates in Human Neuronal CellsDaniel R Whiten, Yukun Zuo, Laura Calo, et al.Cell Reports|December 16, 2017
Inhibiting the Ca2+ Influx Induced by Human CSFAnna Drews, Suman De, Patrick Flagmeier, et al.Nature Communications|April 6, 2019
Different soluble aggregates of Aβ42 can give rise to cellular toxicity through different mechanismsSuman De, David C Wirthensohn, Patrick Flagmeier, et al.Nature Communications|June 14, 2018
α-synuclein oligomers interact with ATP synthase and open the permeability transition pore in Parkinson's diseaseMarthe H R Ludtmann, Plamena R Angelova, Mathew H Horrocks, et al.Pageof 2