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Journal of the American Society of Nephrology : JASN|December 15, 2010
The development of atypical hemolytic uremic syndrome depends on complement C5Elena Goicoechea de Jorge, Paolo Macor, Danielle Paixão-Cavalcante, et al.Proceedings of the National Academy of Sciences of the United States of America|December 13, 2002
The classical pathway is the dominant complement pathway required for innate immunity to Streptococcus pneumoniae infection in miceJeremy S Brown, Tracy Hussell, Sarah M Gilliland, et al.Lupus Science & Medicine|January 25, 2022
Immune gene expression and functional networks in distinct lupus nephritis classesAlyssa C Gilmore, Hannah R Wilson, Thomas D Cairns, et al.Journal of Immunology (Baltimore, Md. : 1950)|February 23, 2002
C1q deficiency and autoimmunity: the effects of genetic background on disease expressionDaniel A Mitchell, Matthew C Pickering, Joanna Warren, et al.The Journal of Experimental Medicine|May 18, 2005
Available carbon source influences the resistance of Neisseria meningitidis against complementRachel M Exley, Jonathan Shaw, Eva Mowe, et al.Arthritis Research & Therapy|April 12, 2006
The in vivo expression of actin/salt-resistant hyperactive DNase I inhibits the development of anti-ssDNA and anti-histone autoantibodies in a murine model of systemic lupus erythematosusAnthony P Manderson, Francesco Carlucci, Peter J Lachmann, et al.Infection and Immunity|July 8, 2009
Complement C3 plays an essential role in the control of opportunistic fungal infectionsS Vicky Tsoni, Ann M Kerrigan, Mohlopheni J Marakalala, et al.Elife|August 11, 2023
TLR7 activation at epithelial barriers promotes emergency myelopoiesis and lung antiviral immunityWilliam D Jackson, Chiara Giacomassi, Sophie Ward, et al.Journal of Leukocyte Biology|May 22, 2009
Roles for phagocytic cells and complement in controlling relapsing fever infectionMichael E Woodman, Anne E Cooley, Rita Avdiushko, et al.Infection and Immunity|June 11, 2008
Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathwayJose Yuste, Ashwin Sen, Lennart Truedsson, et al.Pageof 14