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Innere Medizin (Heidelberg, Germany)|August 5, 2025
[Nephrotic syndrome]Daniela Anne Braun, Hermann-Josef PavenstädtCells|July 11, 2025
A Cell Biologist's View on APOL1: What We Know and What We Still Need to AddressVerena Höffken, Daniela Anne Braun, Hermann Pavenstädt, et al.Life Science Alliance|December 22, 2022
Loss of surface transport is a main cellular pathomechanism of CRB2 variants causing podocytopathiesAnnika Möller-Kerutt, Birgit Schönhoff, Yvonne Rellmann, et al.BMC Genomic Data|November 29, 2024
PALS1-dependent modulations of mRNA profiles in MDCK II cells grown in non-confluent monolayers and three-dimensional cystsKlaus Schughart, Annika Möller-Kerutt, Verena Höffken, et al.Frontiers in Immunology|July 31, 2024
Unveiling systemic responses in kidney transplantation: interplay between the allograft transcriptome and serum proteinsKonrad Buscher, Rebecca Rixen, Paula Schütz, et al.Clinical Journal of the American Society of Nephrology : CJASN|January 21, 2016
Prevalence of Monogenic Causes in Pediatric Patients with Nephrolithiasis or NephrocalcinosisDaniela Anne Braun, Jennifer Ashley Lawson, Heon Yung Gee, et al.Journal of the American Society of Nephrology : JASN|April 6, 2023
Disruption of the Rab7-Dependent Final Common Pathway of Endosomal and Autophagic Processing Results in a Severe PodocytopathyKristin Vöing, Ulf Michgehl, Nils David Mertens, et al.Pageof 1